Nuclear factor-kappaB: a main regulator of inflammation and cell survival in endometriosis pathophysiology.
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Pre-diagnostic serum levels of inflammation markers and risk of ovarian cancer in the prostate, lung, colorectal and ovarian cancer (PLCO) screening trial.Association of the neutrophil-to-lymphocyte ratio and CA 125 with the endometriosis score.Antagonism of betulinic acid on LPS-mediated inhibition of ABCA1 and cholesterol efflux through inhibiting nuclear factor-kappaB signaling pathway and miR-33 expression.Anti-inflammatory, antiangiogenic, and apoptosis-inducing activity of DLBS1442, a bioactive fraction of Phaleria macrocarpa, in a RL95-2 cell line as a molecular model of endometriosis.Naotaifang extract treatment results in increased ferroportin expression in the hippocampus of rats subjected to cerebral ischemia.Pathophysiology of Endometriosis: Role of High Mobility Group Box-1 and Toll-Like Receptor 4 Developing Inflammation in Endometrium.Indoleamine 2,3-dioxygenase-1 (IDO1) enhances survival and invasiveness of endometrial stromal cells via the activation of JNK signaling pathway.Dual suppression of estrogenic and inflammatory activities for targeting of endometriosisIncreased expression of nuclear factor kappa-B p65 subunit in adenomyosis.The endometriotic tissue lining the internal surface of endometrioma: hormonal, genetic, epigenetic status, and gene expression profile.Cyr61 participates in the pathogenesis of acute lymphoblastic leukemia by enhancing cellular survival via the AKT/NF-κB signaling pathway.Possible involvement of CD10 in the development of endometriosis due to its inhibitory effects on CD44-dependent cell adhesion.Risk of ovarian cancer and the NF-κB pathway: genetic association with IL1A and TNFSF10Stem Cell Markers Describe a Transition From Somatic to Pluripotent Cell States in a Rat Model of Endometriosis.The differential expression of mRNAs and long noncoding RNAs between ectopic and eutopic endometria provides new insights into adenomyosis.The P2X7 Receptor Involved in gp120-Induced Cell Injury in BV2 Microglia.Integrative Analysis Reveals Regulatory Programs in Endometriosis.Niclosamide As a Potential Nonsteroidal Therapy for Endometriosis That Preserves Reproductive Function in an Experimental Mouse Model.Mesenchymal Stromal Cells Are More Immunosuppressive In Vitro If They Are Derived from Endometriotic Lesions than from Eutopic Endometrium.Inhibition of IAP (inhibitor of apoptosis) proteins represses inflammatory status via nuclear factor-kappa B pathway in murine endometriosis lesions.Decoy receptor 3 promotes cell adhesion and enhances endometriosis development.Lipopolysaccharide promotes the development of murine endometriosis-like lesions via the nuclear factor-kappa B pathway.Possible Role of α1-Antitrypsin in Endometriosis-Like Grafts From a Mouse Model of Endometriosis.TLR4 Activation Promotes the Secretion of IL-8 Which Enhances the Invasion and Proliferation of Endometrial Stromal Cells in an Autocrine Manner via the FAK Signal Pathway.Shaofu Zhuyu Decoction Regresses Endometriotic Lesions in a Rat Model.Chronic Niche Inflammation in Endometriosis-Associated Infertility: Current Understanding and Future Therapeutic StrategiesAnti-Angiogenic Alternative and Complementary Medicines for the Treatment of Endometriosis: A Review of Potential Molecular Mechanisms
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Nuclear factor-kappaB: a main regulator of inflammation and cell survival in endometriosis pathophysiology.
description
article científic
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article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
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artikull shkencor
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artículo científico
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name
Nuclear factor-kappaB: a main ...... endometriosis pathophysiology.
@en
type
label
Nuclear factor-kappaB: a main ...... endometriosis pathophysiology.
@en
prefLabel
Nuclear factor-kappaB: a main ...... endometriosis pathophysiology.
@en
P2093
P1476
Nuclear factor-kappaB: a main ...... endometriosis pathophysiology.
@en
P2093
Luigi Devoto
Reinaldo González-Ramos
Sylvie Defrère
P304
P356
10.1016/J.FERTNSTERT.2012.06.021
P407
P577
2012-07-06T00:00:00Z