Rationale for combining glutamatergic and cholinergic approaches in the symptomatic treatment of Alzheimer's disease.
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Neuroprotective and symptomatic effects of targeting group III mGlu receptors in neurodegenerative diseaseElevated mRNA-levels of gonadotropin-releasing hormone and its receptor in plaque-bearing Alzheimer's disease transgenic miceConjugates of γ-Carbolines and Phenothiazine as new selective inhibitors of butyrylcholinesterase and blockers of NMDA receptors for Alzheimer DiseaseNeuronal AChE splice variants and their non-hydrolytic functions: redefining a target of AChE inhibitors?Benefits and risks of add-on therapies for Alzheimer's disease.Intrahippocampal Administration of Ibotenic Acid Induced Cholinergic Dysfunction via NR2A/NR2B Expression: Implications of Resveratrol against Alzheimer Disease PathophysiologyRetinal changes in the Tg-SwDI mouse model of Alzheimer's disease.Altered functional connectivity of the default mode network by glucose loading in young, healthy participants.Effects of Tea Catechins on Alzheimer's Disease: Recent Updates and Perspectives
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P2860
Rationale for combining glutamatergic and cholinergic approaches in the symptomatic treatment of Alzheimer's disease.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Rationale for combining glutam ...... atment of Alzheimer's disease.
@en
type
label
Rationale for combining glutam ...... atment of Alzheimer's disease.
@en
prefLabel
Rationale for combining glutam ...... atment of Alzheimer's disease.
@en
P2093
P2860
P356
P1476
Rationale for combining glutam ...... atment of Alzheimer's disease.
@en
P2093
Chris G Parsons
Paul T Francis
Roy W Jones
P2860
P304
P356
10.1586/ERN.12.124
P577
2012-11-01T00:00:00Z