Protein truncation as a common denominator of human neurodegenerative foldopathies.

Protein truncation as a common denominator of human neurodegenerative foldopathies.

Item
http://www.wikidata.org/entity/Q38091670
Decreased O-linked GlcNAcylation protects from cytotoxicity mediated by huntingtin exon1 protein fragmentErythropoietin Modulates Cerebral and Serum Degradation Products from Excess Calpain Activation following Prenatal Hypoxia-Ischemia.Identification of structural determinants on tau protein essential for its pathological function: novel therapeutic target for tau immunotherapy in Alzheimer's disease.Digested disorder, Quarterly intrinsic disorder digest (October-November-December, 2013).Human Truncated Tau Induces Mature Neurofibrillary Pathology in a Mouse Model of Human Tauopathy.Neuronal Expression of Truncated Tau Efficiently Promotes Neurodegeneration in Animal Models: Pitfalls of Toxic Oligomer Analysis.BDNF inhibits neurodegenerative disease-associated asparaginyl endopeptidase activity via phosphorylation by AKT
P2860

Protein truncation as a common denominator of human neurodegenerative foldopathies.

Item
http://www.wikidata.org/entity/Q38091670
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
Protein truncation as a common denominator of human neurodegenerative foldopathies.
@en
type
label
Protein truncation as a common denominator of human neurodegenerative foldopathies.
@en
prefLabel
Protein truncation as a common denominator of human neurodegenerative foldopathies.
@en
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P433
P478
P577
P5875
P6179
P698
P921
P356
P1433
P1476
Protein truncation as a common denominator of human neurodegenerative foldopathies.
@en
P2093
P2860
P2888
P304
P31
P356
P433
P478
P577
P5875
P6179
P698
P921