Understanding the relationship between GSK-3 and Alzheimer's disease: a focus on how GSK-3 can modulate synaptic plasticity processes.
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Protein phosphatase 2A dysfunction in Alzheimer's diseaseIntraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K(+) current inhibition mediated by activation of caspases and GSK-3.SK3 Channel Overexpression in Mice Causes Hippocampal Shrinkage Associated with Cognitive Impairments.Non-Catalytic Roles of Presenilin Throughout Evolution.New insights into the role of glycogen synthase kinase-3 in Alzheimer's disease.Direct High Affinity Interaction between Aβ42 and GSK3α Stimulates Hyperphosphorylation of Tau. A New Molecular Link in Alzheimer's Disease?Haloperidol inactivates AMPK and reduces tau phosphorylation in a tau mouse model of Alzheimer's diseaseAdiponectin Attenuates Streptozotocin-Induced Tau Hyperphosphorylation and Cognitive Deficits by Rescuing PI3K/Akt/GSK-3β Pathway.Peripheral Administration of GSK-3β Antisense Oligonucleotide Improves Learning and Memory in SAMP8 and Tg2576 Mouse Models of Alzheimer's Disease.Bi-directional genetic modulation of GSK-3β exacerbates hippocampal neuropathology in experimental status epilepticus
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Understanding the relationship between GSK-3 and Alzheimer's disease: a focus on how GSK-3 can modulate synaptic plasticity processes.
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article científic
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article scientifique
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articol științific
@ro
articolo scientifico
@it
artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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name
Understanding the relationship ...... synaptic plasticity processes.
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type
label
Understanding the relationship ...... synaptic plasticity processes.
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prefLabel
Understanding the relationship ...... synaptic plasticity processes.
@en
P2860
P356
P1476
Understanding the relationship ...... synaptic plasticity processes.
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P2093
Jesús Avila
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P304
P356
10.1586/ERN.13.39
P577
2013-05-01T00:00:00Z