Neutrophil roles in left ventricular remodeling following myocardial infarction. - Wikidata - wikimedia - TriplyDB

Neutrophil roles in left ventricular remodeling following myocardial infarction.

Neutrophil roles in left ventricular remodeling following myocardial infarction.

http://www.wikidata.org/entity/Q38111517

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The circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarction Stem cell mechanisms during left ventricular remodeling post-myocardial infarction: Repair and regeneration Embolization of the first diagonal branch of the left anterior descending coronary artery as a porcine model of chronic trans-mural myocardial infarction The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is "Inflammation" Always Inflammation?Knowledge gaps to understanding cardiac macrophage polarization following myocardial infarction.Splenic leukocytes mediate the hyperglycemic exacerbation of myocardial infarct size in mice.Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodeling Deficiency of ataxia telangiectasia mutated kinase delays inflammatory response in the heart following myocardial infarction.Avemar and Echinacea extracts enhance mobilization and homing of CD34(+) stem cells in rats with acute myocardial infarction.CD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling.Temporal neutrophil polarization following myocardial infarction Systematic Evaluation of the Cellular Innate Immune Response During the Process of Human Atherosclerosis Matrix metalloproteinase-9: Many shades of function in cardiovascular disease.Endoplasmic reticulum stress in bone marrow-derived cells prevents acute cardiac inflammation and injury in response to angiotensin II.Using systems biology approaches to understand cardiac inflammation and extracellular matrix remodeling in the setting of myocardial infarction Crosstalk between fibroblasts and inflammatory cells.The spatiotemporal cellular dynamics of lung immunity.Cellular immunity and cardiac remodeling after myocardial infarction: role of neutrophils, monocytes, and macrophages.Plasma-derived Extracellular Vesicles Contain Predictive Biomarkers and Potential Therapeutic Targets for Myocardial Ischemic (MI) Injury.Leukocyte diversity in resolving and nonresolving mechanisms of cardiac remodeling.Mechanisms of action of sacubitril/valsartan on cardiac remodeling: a systems biology approach.11β-HSD1 suppresses cardiac fibroblast CXCL2, CXCL5 and neutrophil recruitment to the heart post MI.Modeling the Human Scarred Heart In Vitro: Toward New Tissue Engineered Models.The Role of Cardiac Tissue Macrophages in Homeostasis and Disease.Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.RORγt-expressing cells attenuate cardiac remodeling after myocardial infarction.Optimal Environmental Stiffness for Stem Cell Mediated Ischemic Myocardium Repair.Independent trafficking of flavocytochrome b558 and myeloperoxidase to phagosomes during phagocytosis visualised by energy-filtering and energy-dispersive spectroscopy-scanning transmission electron microscopy.Anatomical-Molecular Distribution of EphrinA1 in Infarcted Mouse Heart Using MALDI Mass Spectrometry Imaging.Inhibition of the Renin-Angiotensin System Post Myocardial Infarction Prevents Inflammation-Associated Acute Cardiac Rupture.Properties and Immune Function of Cardiac Fibroblasts.PKM2 released by neutrophils at wound site facilitates early wound healing by promoting angiogenesis.Neutrophils orchestrate post-myocardial infarction healing by polarizing macrophages towards a reparative phenotype.Molecular imaging of myocardial infarction with Gadofluorine P - A combined magnetic resonance and mass spectrometry imaging approach.Immunomodulatory interventions in myocardial infarction and heart failure: a systematic review of clinical trials and meta-analysis of IL-1 inhibition

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Neutrophil roles in left ventricular remodeling following myocardial infarction.

http://www.wikidata.org/entity/Q38111517

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Neutrophil roles in left ventricular remodeling following myocardial infarction.

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Neutrophil roles in left ventricular remodeling following myocardial infarction.

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Neutrophil roles in left ventricular remodeling following myocardial infarction.

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Fibrogenesis and Tissue Repair

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Neutrophil roles in left ventricular remodeling following myocardial infarction

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Yonggang Ma

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P2860

Human neutrophil defensins selectively chemoattract naive T and immature dendritic cells

In vivo labeling with 2H2O reveals a human neutrophil lifespan of 5.4 days

Secretion of heparin-binding protein from human neutrophils is determined by its localization in azurophilic granules and secretory vesicles

Neutrophil-derived azurocidin cleaves insulin-like growth factor-binding protein-1, -2 and -4

Regulation of neutrophil function by adenosine.

Neutrophil primary granule proteins HBP and HNP1-3 boost bacterial phagocytosis by human and murine macrophages

The solution structure and dynamics of human neutrophil gelatinase-associated lipocalin

Neutrophil extracellular traps kill bacteria

NSP4, an elastase-related protease in human neutrophils with arginine specificity

Neutrophil granules and secretory vesicles in inflammation

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10.1186/1755-1536-6-11

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Merry L. Lindsey

Andriy Yabluchanskiy

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2013-06-03T00:00:00Z

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