Determinants of myocardial conduction velocity: implications for arrhythmogenesis. - Wikidata - wikimedia - TriplyDB

Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

http://www.wikidata.org/entity/Q38119213

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Mechanobiology of myofibroblast adhesion in fibrotic cardiac disease Autonomous beating rate adaptation in human stem cell-derived cardiomyocytes.Caveolae in ventricular myocytes are required for stretch-dependent conduction slowing Caveolin-1 modulates cardiac gap junction homeostasis and arrhythmogenecity by regulating cSrc tyrosine kinase.Sodium channel biophysics, late sodium current and genetic arrhythmic syndromes Cdon deficiency causes cardiac remodeling through hyperactivation of WNT/β-catenin signaling Techniques for automated local activation time annotation and conduction velocity estimation in cardiac mapping.Association of Left Atrial Local Conduction Velocity With Late Gadolinium Enhancement on Cardiac Magnetic Resonance in Patients With Atrial Fibrillation.The RyR2-P2328S mutation downregulates Nav1.5 producing arrhythmic substrate in murine ventricles.Cardiac sodium channel mutations: why so many phenotypes?The effect of electrical conductivity of myocardium on cardiac pumping efficacy: a computational study.Republished: A straightforward guide to the basic science behind arrhythmogenesis.The Impact of Cardiac Radiation Dosimetry on Survival After Radiation Therapy for Non-Small Cell Lung Cancer.Transient Notch Activation Induces Long-Term Gene Expression Changes Leading to Sick Sinus Syndrome in Mice.Mathematical modeling of cardiac growth and remodeling.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Role of abnormal repolarization in the mechanism of cardiac arrhythmia.Type 2 Diabetes Induces Prolonged P-wave Duration without Left Atrial Enlargement Atrial electrocardiography in obesity and hypertension: Clinical insights from the Polish-Norwegian Study (PONS).Evaluation of Index of Cardio-Electrophysiological Balance (iCEB) as a New Biomarker for the Identification of Patients at Increased Arrhythmic Risk.Illuminating Myocyte-Fibroblast Homotypic and Heterotypic Gap Junction Dynamics Using Dynamic Clamp.The effects of ageing and adrenergic challenge on electrocardiographic phenotypes in a murine model of long QT syndrome type 3 De Novo FGF12 (Fibroblast Growth Factor 12) Functional Variation Is Potentially Associated With Idiopathic Ventricular Tachycardia.Sudden arrhythmic death: from basic science to clinical practice Flecainide exerts paradoxical effects on sodium currents and atrial arrhythmia in murine RyR2-P2328S hearts.Functional dissection of myosin binding protein C phosphorylation.Through modulation of cardiac Ca2+ handling, UCP2 affects cardiac electrophysiology and influences the susceptibility for Ca2+ -mediated arrhythmias.Age-dependent atrial arrhythmic phenotype secondary to mitochondrial dysfunction in Pgc-1β deficient murine hearts.The effect of activation rate on left atrial bipolar voltage in patients with paroxysmal atrial fibrillation.Pro-arrhythmic atrial phenotypes in incrementally paced murine Pgc1β-/- hearts: effects of age.Incorporating inductances in tissue-scale models of cardiac electrophysiology.Epac-induced ryanodine receptor type 2 activation inhibits sodium currents in atrial and ventricular murine cardiomyocytes.A mutant of the Buthus martensii Karsch antitumor-analgesic peptide exhibits reduced inhibition to hNav1.4 and hNav1.5 channels while retaining analgesic activity.Uniaxial strain of cultured mouse and rat cardiomyocyte strands slows conduction more when its axis is parallel to impulse propagation than when it is perpendicular.Cardiomyocyte ionic currents in intact young and aged murine Pgc-1β-/- atrial preparations.Ventricular pro-arrhythmic phenotype, arrhythmic substrate, ageing and mitochondrial dysfunction in peroxisome proliferator activated receptor-γ coactivator-1β deficient (Pgc-1β-/-) murine hearts.ETV1 activates a rapid conduction transcriptional program in rodent and human cardiomyocytes.Complex interactions in a novel SCN5A compound mutation associated with long QT and Brugada syndrome: Implications for Na+ channel blocking pharmacotherapy for de novo conduction disease.Computational Modeling of Electrophysiology and Pharmacotherapy of Atrial Fibrillation: Recent Advances and Future Challenges Muscle Thickness and Curvature Influence Atrial Conduction Velocities

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Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

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Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

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Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

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Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

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FPHYS.2013.00154

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Frontiers in Physiology

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Determinants of myocardial conduction velocity: implications for arrhythmogenesis.

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Christopher L-H Huang

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James A Fraser

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James H King

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P2860

Cardiac conduction defects associate with mutations in SCN5A

The effect of sodium ions on the electrical activity of the giant axon of the squid

Somatic mutations in the connexin 40 gene (GJA5) in atrial fibrillation

Mutations of the Connexin43 gap-junction gene in patients with heart malformations and defects of laterality

Solution NMR structure of the C-terminal EF-hand domain of human cardiac sodium channel NaV1.5

Primary structure and functional expression of the human cardiac tetrodotoxin-insensitive voltage-dependent sodium channel

Connexin43: a protein from rat heart homologous to a gap junction protein from liver

A single Na(+) channel mutation causing both long-QT and Brugada syndromes

A sodium-channel mutation causes isolated cardiac conduction disease

Congenital sick sinus syndrome caused by recessive mutations in the cardiac sodium channel gene (SCN5A)

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