Current status of NADPH oxidase research in cardiovascular pharmacology - Wikidata - wikimedia - TriplyDB

Current status of NADPH oxidase research in cardiovascular pharmacology

Current status of NADPH oxidase research in cardiovascular pharmacology

http://www.wikidata.org/entity/Q38132120

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Are Endothelial Progenitor Cells the Real Solution for Cardiovascular Diseases? Focus on Controversies and Perspectives Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2 Elevated peripheral blood mononuclear cell-derived superoxide production in healthy young black men.CD38 mediates angiotensin II-induced intracellular Ca(2+) release in rat pulmonary arterial smooth muscle cells.Nox Inhibitors & Therapies: Rational Design of Peptidic and Small Molecule Inhibitors Impaired compensation to femoral artery ligation in diet-induced obese mice is primarily mediated via suppression of collateral growth by Nox2 and p47phox.Selective inactivation of NADPH oxidase 2 causes regression of vascularization and the size and stability of atherosclerotic plaques NOX4 downregulation leads to senescence of human vascular smooth muscle cells.The Importance of NADPH Oxidases and Redox Signaling in Angiogenesis.Cholesterol: A modulator of the phagocyte NADPH oxidase activity - A cell-free study.Anesthetic-induced developmental neurotoxicity: causes, prospective studies and possible interventions.Scavenging of nitric oxide by hemoglobin in the tunica media of porcine coronary arteries.

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Current status of NADPH oxidase research in cardiovascular pharmacology

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Vascular Health and Risk Management

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Current status of NADPH oxidase research in cardiovascular pharmacology

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Beatriz Paradela-Dobarro

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Ezequiel Alvarez

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María Isabel Castiñeiras-Landeira

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P2860

A Ca(2+)-activated NADPH oxidase in testis, spleen, and lymph nodes

NOX5 variants are functionally active in endothelial cells

Anion channels, including ClC-3, are required for normal neutrophil oxidative function, phagocytosis, and transendothelial migration

Distinct roles of Nox1 and Nox4 in basal and angiotensin II-stimulated superoxide and hydrogen peroxide production

Cytokine activation of nuclear factor kappa B in vascular smooth muscle cells requires signaling endosomes containing Nox1 and ClC-3

VE-PTP and VE-cadherin ectodomains interact to facilitate regulation of phosphorylation and cell contacts

IQGAP proteins are integral components of cytoskeletal regulation

NAD(P)H oxidase Nox-4 mediates 7-ketocholesterol-induced endoplasmic reticulum stress and apoptosis in human aortic smooth muscle cells

Efficacy of vitamin and antioxidant supplements in prevention of cardiovascular disease: systematic review and meta-analysis of randomised controlled trials

Targeting NADPH oxidases in vascular pharmacology

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401-428

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10.2147/VHRM.S33053

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9

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Sergio Raposeiras-Roubín

Bruno K Rodiño-Janeiro

José R González-Juanatey

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2013-07-25T00:00:00Z

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256190118

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23983473

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inorganic compound

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cardiovascular disease

membrane proteins

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3750863

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