Transforming growth factor-β and the progression of renal disease.
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Emerging Transcriptional Mechanisms in the Regulation of Epithelial to Mesenchymal Transition and Cellular Plasticity in the KidneyEpithelial-to-Mesenchymal Transition in Diabetic Nephropathy: Fact or Fiction?Cellular and Molecular Mechanisms of Chronic Kidney Disease with Diabetes Mellitus and Cardiovascular Diseases as Its ComorbiditiesTGF-β/Smad signaling in renal fibrosisAblation of the N-type calcium channel ameliorates diabetic nephropathy with improved glycemic control and reduced blood pressure.Impact of heparanase on renal fibrosisBrd4 inhibition attenuates unilateral ureteral obstruction-induced fibrosis by blocking TGF-β-mediated Nox4 expressionSignalling pathways involved in hypoxia-induced renal fibrosis.Epoxyeicosatrienoic Acid Analog Decreases Renal Fibrosis by Reducing Epithelial-to-Mesenchymal Transition.From molecular signatures to predictive biomarkers: modeling disease pathophysiology and drug mechanism of action.MicroRNA-328 inhibits renal tubular cell epithelial-to-mesenchymal transition by targeting the CD44 in pressure-induced renal fibrosis.Stem cell conditioned culture media attenuated albumin-induced epithelial-mesenchymal transition in renal tubular cells.Profiling and initial validation of urinary microRNAs as biomarkers in IgA nephropathyAllelic Variants in Arhgef11 via the Rho-Rock Pathway Are Linked to Epithelial-Mesenchymal Transition and Contributes to Kidney Injury in the Dahl Salt-Sensitive RatSimultaneous deletion of Bax and Bak is required to prevent apoptosis and interstitial fibrosis in obstructive nephropathyTwenty years after ACEIs and ARBs: emerging treatment strategies for diabetic nephropathy.Epigenetic Histone Modifications Involved in Profibrotic Gene Regulation by 12/15-Lipoxygenase and Its Oxidized Lipid Products in Diabetic Nephropathymyo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience: A NEW MECHANISM RELEVANT TO THE PATHOGENESIS OF DIABETIC NEPHROPATHYInsights into the Mechanisms Involved in the Expression and Regulation of Extracellular Matrix Proteins in Diabetic Nephropathy.Transforming growth factor β1 antagonizes the transcription, expression and vascular signaling of guanylyl cyclase/natriuretic peptide receptor A - role of δEF1.Enhancer of Zeste Homolog 2 Inhibition Attenuates Renal Fibrosis by Maintaining Smad7 and Phosphatase and Tensin Homolog ExpressionThe Hippo-Salvador signaling pathway regulates renal tubulointerstitial fibrosis.Effects of transforming growth factor β2 and connective tissue growth factor on induction of epithelial mesenchymal transition and extracellular matrix synthesis in human lens epithelial cells.PHF14: an innate inhibitor against the progression of renal fibrosis following folic acid-induced kidney injury.Inflammatory biomarkers in type 2 diabetic patients: effect of glycemic control and impact of LDL subfraction phenotype.Oligo-fucoidan prevents renal tubulointerstitial fibrosis by inhibiting the CD44 signal pathway.Nicousamide protects kidney podocyte by inhibiting the TGFβ receptor II phosphorylation and AGE-RAGE signalingDiabetes-Induced Reactive Oxygen Species: Mechanism of Their Generation and Role in Renal InjuryMicroRNAs in IgA nephropathy.The role of endoglin in kidney fibrosis.Long-term sequelae from acute kidney injury: potential mechanisms for the observed poor renal outcomes.How tubular epithelial cells dictate the rate of renal fibrogenesis?Glomerular cell crosstalk.Connexin43 Controls the Myofibroblastic Differentiation of Bronchial Fibroblasts from Patients with Asthma.Signaling Crosstalk between Tubular Epithelial Cells and Interstitial Fibroblasts after Kidney Injury.Beneficial Effect of Short Pretransplant Period of Hypothermic Pulsatile Perfusion of the Warm-Ischemic Kidney after Cold Storage: Experimental Study.Recombinant fibromodulin has therapeutic effects on diabetic nephropathy by down-regulating transforming growth factor-β1 in streptozotocin-induced diabetic rat model.Urine matrix metalloproteinases and their extracellular inducer EMMPRIN in children with chronic kidney disease.The Use of Cytochrome C Oxidase Enzyme Activity and Immunohistochemistry in Defining Mitochondrial Injury in Kidney DiseaseRho-Kinase Blockade Attenuates Podocyte Apoptosis by Inhibiting the Notch Signaling Pathway in Diabetic Nephropathy
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Transforming growth factor-β and the progression of renal disease.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
Transforming growth factor-β and the progression of renal disease.
@en
type
label
Transforming growth factor-β and the progression of renal disease.
@en
prefLabel
Transforming growth factor-β and the progression of renal disease.
@en
P356
P1476
Transforming growth factor-β and the progression of renal disease.
@en
P2093
Gunter Wolf
Ivonne Loeffler
P304
P356
10.1093/NDT/GFT267
P407
P478
29 Suppl 1
P577
2013-09-12T00:00:00Z