Roles of changes in active glutamine transport in brain edema development during hepatic encephalopathy: an emerging concept
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Advances in urea cycle neuroimaging: Proceedings from the 4th International Symposium on urea cycle disorders, Barcelona, Spain, September 2013In vivo N-15 MRS study of glutamate metabolism in the rat brain.Neuronal gamma-aminobutyric acid (GABA) type A receptors undergo cognate ligand chaperoning in the endoplasmic reticulum by endogenous GABA.Ammonia, like K(+), stimulates the Na(+), K(+), 2 Cl(-) cotransporter NKCC1 and the Na(+),K(+)-ATPase and interacts with endogenous ouabain in astrocytes.Multifactorial Effects on Different Types of Brain Cells Contribute to Ammonia Toxicity.Solute Carriers in the Blood-Brain Barier: Safety in Abundance.A neuronal disruption in redox homeostasis elicited by ammonia alters the glycine/glutamate (GABA) cycle and contributes to MMA-induced excitability.Glucose, Lactate, β-Hydroxybutyrate, Acetate, GABA, and Succinate as Substrates for Synthesis of Glutamate and GABA in the Glutamine-Glutamate/GABA Cycle.System N transporters are critical for glutamine release and modulate metabolic fluxes of glucose and acetate in cultured cortical astrocytes: changes induced by ammonia.
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Roles of changes in active glutamine transport in brain edema development during hepatic encephalopathy: an emerging concept
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artículo científico
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Roles of changes in active glu ...... halopathy: an emerging concept
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Roles of changes in active glu ...... halopathy: an emerging concept
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prefLabel
Roles of changes in active glu ...... halopathy: an emerging concept
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Roles of changes in active glu ...... halopathy: an emerging concept
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Jan Albrecht
Magdalena Zielińska
Mariusz Popek
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P356
10.1007/S11064-013-1141-X
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2013-09-26T00:00:00Z
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P6179
1000149343