Adverse fibrosis in the aging heart depends on signaling between myeloid and mesenchymal cells; role of inflammatory fibroblasts.
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Age-associated pro-inflammatory remodeling and functional phenotype in the heart and large arteriesSuppression of Transforming Growth Factor-β Signaling Delays Cellular Senescence and Preserves the Function of Endothelial Cells Derived from Human Pluripotent Stem Cells.Influences of age-related changes in mesenchymal stem cells on macrophages during in-vitro culture.Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease.Aging disrupts cell subpopulation dynamics and diminishes the function of mesenchymal stem cells.Transition of mesothelial cell to fibroblast in peritoneal dialysis: EMT, stem cell or bystander?Tumor necrosis factor: a mechanistic link between angiotensin-II-induced cardiac inflammation and fibrosis.Adult vascular wall resident multipotent vascular stem cells, matrix metalloproteinases, and arterial aneurysms.Mesenchymal stem cell-derived inflammatory fibroblasts promote monocyte transition into myeloid fibroblasts via an IL-6-dependent mechanism in the aging mouse heart.Role of Circulating Fibrocytes in Cardiac FibrosisInternal associations and dynamic expression of c-kit and nanog genes in ventricular remodelling induced by adriamycinThe role of extracellular matrix in age-related conduction disorders: a forgotten player?Cardiac Physiology of Aging: Extracellular Considerations.Mesenchymal stem cell-derived inflammatory fibroblasts mediate interstitial fibrosis in the aging heart.Aicar treatment reduces interstitial fibrosis in aging mice: Suppression of the inflammatory fibroblast.Can heart function lost to disease be regenerated by therapeutic targeting of cardiac scar tissue?Controlled Inhibition of the Mesenchymal Stromal Cell Pro-inflammatory Secretome via Microparticle Engineering.CD45 Expression in Mitral Valve Endothelial Cells After Myocardial Infarction.Bone marrow and adipose mesenchymal stem cells attenuate cardiac fibrosis induced by methotrexate in rats.The allosteric glycogen synthase kinase-3 inhibitor NP12 limits myocardial remodeling and promotes angiogenesis in an acute myocardial infarction model.Dissecting the role of myeloid and mesenchymal fibroblasts in age-dependent cardiac fibrosis.Advanced glycation end-products accelerate the cardiac aging process through the receptor for advanced glycation end-products/transforming growth factor-β-Smad signaling pathway in cardiac fibroblasts.Reversal of maladaptive fibrosis and compromised ventricular function in the pressure overloaded heart by a caveolin-1 surrogate peptide.Sirtuins and Accelerated Aging in Scleroderma.Improved Cardiovascular Function in Old Mice after N-Acetyl Cysteine and Glycine Supplemented Diet: Inflammation and Mitochondrial Factors.
P2860
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P2860
Adverse fibrosis in the aging heart depends on signaling between myeloid and mesenchymal cells; role of inflammatory fibroblasts.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artículo científico
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name
Adverse fibrosis in the aging ...... e of inflammatory fibroblasts.
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type
label
Adverse fibrosis in the aging ...... e of inflammatory fibroblasts.
@en
prefLabel
Adverse fibrosis in the aging ...... e of inflammatory fibroblasts.
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P2093
P2860
P1476
Adverse fibrosis in the aging ...... e of inflammatory fibroblasts.
@en
P2093
George E Taffet
Jeffrey R Crawford
JoAnn Trial
Katarzyna A Cieslik
Mark L Entman
P2860
P356
10.1016/J.YJMCC.2013.10.017
P577
2013-10-31T00:00:00Z