Molecular and cellular mechanism of okadaic acid (OKA)-induced neurotoxicity: a novel tool for Alzheimer's disease therapeutic application. - Wikidata - wikimedia - TriplyDB

Molecular and cellular mechanism of okadaic acid (OKA)-induced neurotoxicity: a novel tool for Alzheimer's disease therapeutic application.

Molecular and cellular mechanism of okadaic acid (OKA)-induced neurotoxicity: a novel tool for Alzheimer's disease therapeutic application.

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Natural Phytochemicals in the Treatment and Prevention of Dementia: An Overview Rodent models of neuroinflammation for Alzheimer's disease Tau physiology and pathomechanisms in frontotemporal lobar degeneration Marine-Derived Pharmaceuticals - Challenges and Opportunities Bryostatin-1 Restores Blood Brain Barrier Integrity following Blast-Induced Traumatic Brain Injury PP2A inhibition from LB100 therapy enhances daunorubicin cytotoxicity in secondary acute myeloid leukemia via miR-181b-1 upregulation.Astrocyte mediated MMP-9 activation in the synapse dysfunction: An implication in Alzheimer disease.A new coumarin derivative, IMM-H004, attenuates okadaic acid-induced spatial memory impairment in rats Original Research: Influence of okadaic acid on hyperphosphorylation of tau and nicotinic acetylcholine receptors in primary neurons Inhibition of Protein Phosphatase 2A: Focus on the Glutamatergic System.Sulforaphane Ameliorates Okadaic Acid-Induced Memory Impairment in Rats by Activating the Nrf2/HO-1 Antioxidant Pathway.Protein Phosphatase 2A: a Double-Faced Phosphatase of Cellular System and Its Role in Neurodegenerative Disorders.Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.Utilizing zebrafish and okadaic acid to study Alzheimer's disease

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Molecular and cellular mechanism of okadaic acid (OKA)-induced neurotoxicity: a novel tool for Alzheimer's disease therapeutic application.

http://www.wikidata.org/entity/Q38202889

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Molecular and cellular mechani ...... sease therapeutic application.

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Molecular and cellular mechani ...... sease therapeutic application.

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S12035-014-8699-4

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Molecular Neurobiology

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Molecular and cellular mechani ...... sease therapeutic application.

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Chandishwar Nath

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Pradip K Kamat

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Rakesh Shukla

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Shivika Rai

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Supriya Swarnkar

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P2860

Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

Signal transduction by the JNK group of MAP kinases

Neuronal CA2+/calmodulin-dependent protein kinase II: the role of structure and autoregulation in cellular function

Ras activation of the Raf kinase: tyrosine kinase recruitment of the MAP kinase cascade

Ca(2+)/CaM-dependent kinases: from activation to function

Neuronal Ca2+/calmodulin-dependent protein kinase II--discovery, progress in a quarter of a century, and perspective: implication for learning and memory

Reconsider Alzheimer's disease by the 'calpain-cathepsin hypothesis'--a perspective review

GSK-3beta is required for memory reconsolidation in adult brain

Mediators of endoplasmic reticulum stress-induced apoptosis

Neurodegenerative tauopathies

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s12035-014-8699-4

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852-865

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10.1007/S12035-014-8699-4

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3

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50

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2014-04-08T00:00:00Z

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24710687

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Alzheimer's disease