The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases. - Wikidata - wikimedia - TriplyDB

The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases.

The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases.

http://www.wikidata.org/entity/Q38203518

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Nitro-Oleic Acid Prevents Hypoxia- and Asymmetric Dimethylarginine-Induced Pulmonary Endothelial Dysfunction.Plasma levels of dimethylarginines in preterm very low birth weight neonates: its relation with perinatal factors and short-term outcome.Redox Control of Protein Arginine Methyltransferase 1 (PRMT1) Activity.Long-Term Chronic Intermittent Hypobaric Hypoxia in Rats Causes an Imbalance in the Asymmetric Dimethylarginine/Nitric Oxide Pathway and ROS Activity: A Possible Synergistic Mechanism for Altitude Pulmonary Hypertension?Endogenous Asymmetric Dimethylarginine Pathway in High Altitude Adapted Yaks.L-Arginine Pathway in COPD Patients with Acute Exacerbation: A New Potential Biomarker.Relationship of asymmetric dimethylarginine levels with disease severity and pulmonary hypertension in chronic obstructive pulmonary disease.

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The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases.

http://www.wikidata.org/entity/Q38203518

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article científic

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The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases.

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The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases.

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The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases.

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BioMed Research International

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The endothelial ADMA/NO pathway in hypoxia-related chronic respiratory diseases

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Nicole Lüneburg

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Creative Commons Attribution 3.0 Unported

P2860

HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing

C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

Studies on the reaction of nitric oxide with the hypoxia-inducible factor prolyl hydroxylase domain 2 (EGLN1)

The L-arginine-nitric oxide pathway

S-nitrosylation of dimethylarginine dimethylaminohydrolase regulates enzyme activity: further interactions between nitric oxide synthase and dimethylarginine dimethylaminohydrolase

Cloned and expressed macrophage nitric oxide synthase contrasts with the brain enzyme

Molecular pathogenesis of pulmonary arterial hypertension

S-nitrosothiols signal hypoxia-mimetic vascular pathology

Epithelial inducible nitric oxide synthase activity is the major determinant of nitric oxide concentration in exhaled breath

Nitric oxide: physiology, pathophysiology, and pharmacology

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10.1155/2014/501612

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2014

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2014-02-25T00:00:00Z

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