about
Discovery and prioritization of somatic mutations in diffuse large B-cell lymphoma (DLBCL) by whole-exome sequencingRisk of non-Hodgkin lymphoma in association with germline variation in complement genesBone marrow stromal cells protect lymphoma B-cells from rituximab-induced apoptosis and targeting integrin α-4-β-1 (VLA-4) with natalizumab can overcome this resistanceGenome-wide association study identifies multiple susceptibility loci for diffuse large B cell lymphomaGenetically predicted longer telomere length is associated with increased risk of B-cell lymphoma subtypesGWAS of follicular lymphoma reveals allelic heterogeneity at 6p21.32 and suggests shared genetic susceptibility with diffuse large B-cell lymphomaGenome-wide association study identifies five susceptibility loci for follicular lymphoma outside the HLA region.Germline variation in apoptosis pathway genes and risk of non-Hodgkin's lymphoma.A genome-wide association study of marginal zone lymphoma shows association to the HLA region.Non-Hodgkin Lymphoma, Body Mass Index, and Cytokine Polymorphisms: A Pooled Analysis from the InterLymph Consortium.A two-stage evaluation of genetic variation in immune and inflammation genes with risk of non-Hodgkin lymphoma identifies new susceptibility locus in 6p21.3 region.PRRC2A and BCL2L11 gene variants influence risk of non-Hodgkin lymphoma: results from the InterLymph consortium.Germline variation in complement genes and event-free survival in follicular and diffuse large B-cell lymphoma.Meta-analysis of genome-wide association studies discovers multiple loci for chronic lymphocytic leukemia.Mapping of the IRF8 gene identifies a 3'UTR variant associated with risk of chronic lymphocytic leukemia but not other common non-Hodgkin lymphoma subtypes.Common variants within 6p21.31 locus are associated with chronic lymphocytic leukaemia and, potentially, other non-Hodgkin lymphoma subtypes.Genome-Wide Association Study of Event-Free Survival in Diffuse Large B-Cell Lymphoma Treated With Immunochemotherapy.CXCR5 polymorphisms in non-Hodgkin lymphoma risk and prognosis.A proliferation-inducing ligand mediates follicular lymphoma B-cell proliferation and cyclin D1 expression through phosphatidylinositol 3-kinase-regulated mammalian target of rapamycin activationGenetic variation in tumor necrosis factor and the nuclear factor-kappaB canonical pathway and risk of non-Hodgkin's lymphoma.Genetic variation in B-cell-activating factor is associated with an increased risk of developing B-cell non-Hodgkin lymphoma.
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description
forsker ved Mayo Clinic
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hematoloog
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researcher at the Mayo Clinic, hematologist
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name
Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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Anne J. Novak
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P108
P2038
Anne_Novak