Animal models of bronchopulmonary dysplasia. The term rat models.
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Endogenous and Exogenous Stem/Progenitor Cells in the Lung and Their Role in the Pathogenesis and Treatment of Pediatric Lung DiseaseAffect of Early Life Oxygen Exposure on Proper Lung Development and Response to Respiratory Viral InfectionsStem Cells and Their Mediators - Next Generation Therapy for Bronchopulmonary DysplasiaThe impact of vitamin D on fetal and neonatal lung maturation. A systematic reviewJuvenile Toxicology: Relevance and Challenges for Toxicologists and PathologistsCan maternal DHA supplementation offer long-term protection against neonatal hyperoxic lung injury?Optimal Route for Human Umbilical Cord Blood-Derived Mesenchymal Stem Cell Transplantation to Protect Against Neonatal Hyperoxic Lung Injury: Gene Expression Profiles and HistopathologyNeonatal hyperoxic lung injury favorably alters adult right ventricular remodeling response to chronic hypoxia exposureThe H2S-generating enzymes cystathionine β-synthase and cystathionine γ-lyase play a role in vascular development during normal lung alveolarization.Impaired pulmonary vascular development in bronchopulmonary dysplasia.Hyperoxia-Induced Proliferative Retinopathy: Early Interruption of Retinal Vascular Development with Severe and Irreversible Neurovascular Disruption.Antenatal endotoxin disrupts lung vitamin D receptor and 25-hydroxyvitamin D 1α-hydroxylase expression in the developing rat.Protective effect of chorioamnionitis on the development of bronchopulmonary dysplasia triggered by postnatal systemic inflammation in neonatal rats.Impact of bronchopulmonary dysplasia on brain and retina.Adult Lysophosphatidic Acid Receptor 1-Deficient Rats with Hyperoxia-Induced Neonatal Chronic Lung Disease Are Protected against Lipopolysaccharide-Induced Acute Lung Injury.Progressive Vascular Functional and Structural Damage in a Bronchopulmonary Dysplasia Model in Preterm Rabbits Exposed to Hyperoxia.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Aberrant cGMP signaling persists during recovery in mice with oxygen-induced pulmonary hypertension.A new target for caffeine in the developing lung: endoplasmic reticulum stress?An Official American Thoracic Society Workshop Report 2015. Stem Cells and Cell Therapies in Lung Biology and Diseases.Caffeine ameliorates hyperoxia-induced lung injury by protecting GCH1 function in neonatal rat pups.Cell-based therapies for neonatal lung disease.Postnatal Hyperoxia Exposure Durably Impairs Right Ventricular Function and Mitochondrial Biogenesis.Looking ahead: where to next for animal models of bronchopulmonary dysplasia?Bone Morphogenetic Protein 9 Protects against Neonatal Hyperoxia-Induced Impairment of Alveolarization and Pulmonary Inflammation.Collagen and elastin cross-linking is altered during aberrant late lung development associated with hyperoxia.A three-dimensional human model of the fibroblast activation that accompanies bronchopulmonary dysplasia identifies Notch-mediated pathophysiology.Neonatal exposure to high oxygen levels leads to impaired ischemia-induced neovascularization in adulthood.Searching for better animal models of BPD: a perspective.Posttranslational modification of β-catenin is associated with pathogenic fibroblastic changes in bronchopulmonary dysplasia.Hyperoxia-mediated LC3B activation contributes to the impaired transdifferentiation of type II alveolar epithelial cells (AECIIs) to type I cells (AECIs).Phosphatidylcholine kinetics in neonatal rat lungs and the effects of rhuKGF and betamethasone.Hydrogen-rich water ameliorates bronchopulmonary dysplasia (BPD) in newborn rats.Lung microRNA deregulation associated with impaired alveolarization in rats after intrauterine growth restriction.Neonatal hyperoxia depletes pulmonary vein cardiomyocytes in adult mice via mitochondrial oxidation.Recent advances in our understanding of the mechanisms of late lung development and bronchopulmonary dysplasia.Florid Intussusceptive-like Microvascular Dysangiogenesis in a Preterm Lung.Inhibition of IκBβ/NFκB signaling prevents LPS-induced IL1β expression without increasing apoptosis in the developing mouse lung.Attenuation of endoplasmic reticulum stress by caffeine ameliorates hyperoxia-induced lung injury.Postnatal Organ Development as a Complicating Factor in Juvenile Toxicity Studies in Rats.
P2860
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P2860
Animal models of bronchopulmonary dysplasia. The term rat models.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh
2014年學術文章
@zh-hant
name
Animal models of bronchopulmonary dysplasia. The term rat models.
@en
type
label
Animal models of bronchopulmonary dysplasia. The term rat models.
@en
prefLabel
Animal models of bronchopulmonary dysplasia. The term rat models.
@en
P1476
Animal models of bronchopulmonary dysplasia. The term rat models.
@en
P2093
Bernard Thébaud
Megan O'Reilly
P304
P356
10.1152/AJPLUNG.00160.2014
P577
2014-10-10T00:00:00Z