Mutant K-Ras increases GSK-3β gene expression via an ETS-p300 transcriptional complex in pancreatic cancer.
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Poly(ADP-ribose) polymerase inhibitors sensitize cancer cells to death receptor-mediated apoptosis by enhancing death receptor expression.Essential gene profiles in breast, pancreatic, and ovarian cancer cells.High cell surface death receptor expression determines type I versus type II signalingCommon Polymorphisms in the GSK3β Gene May Contribute to the Pathogenesis of Alzheimer Disease: A Meta-Analysis.Trafficking defects in WASH-knockout fibroblasts originate from collapsed endosomal and lysosomal networks.Aberrant glycogen synthase kinase 3β in the development of pancreatic cancer.KRAS gene amplification and overexpression but not mutation associates with aggressive and metastatic endometrial cancer.Chemopreventive effect of resveratrol and apocynin on pancreatic carcinogenesis via modulation of nuclear phosphorylated GSK3β and ERK1/2Upstream and Downstream Co-inhibition of Mitogen-Activated Protein Kinase and PI3K/Akt/mTOR Pathways in Pancreatic Ductal Adenocarcinoma.A cAMP and CREB-mediated feed-forward mechanism regulates GSK3β in polycystic kidney disease.Modulation of glycogen synthase kinase-3β following TRAIL combinatorial treatment in cancer cells.The ubiquitin-proteasome system and signal transduction pathways regulating Epithelial Mesenchymal transition of cancer.Mechanisms of RAS/β-catenin interactions.Glycogen Synthase Kinase 3β in Pancreatic Cancer and its Implications in Chemotherapy and Radiation Therapy.MYC in pancreatic cancer: novel mechanistic insights and their translation into therapeutic strategies.GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression.Differential activity of GSK-3 isoforms regulates NF-κB and TRAIL- or TNFα induced apoptosis in pancreatic cancer cellsFlavonoid apigenin modified gene expression associated with inflammation and cancer and induced apoptosis in human pancreatic cancer cells through inhibition of GSK-3β/NF-κB signaling cascade.GSK-3α promotes oncogenic KRAS function in pancreatic cancer via TAK1-TAB stabilization and regulation of noncanonical NF-κB.Ras Activity Tunes the Period and Modulates the Entrainment of the Suprachiasmatic Clock.The cytoskeletal adaptor protein IQGAP1 regulates TCR-mediated signaling and filamentous actin dynamics.11'-Deoxyverticillin A (C42) promotes autophagy through K-Ras/GSK3 signaling pathway in HCT116 cells.Glycogen synthase kinase-3β ablation limits pancreatitis-induced acinar-to-ductal metaplasia.Ras Activity Oscillates in the Mouse Suprachiasmatic Nucleus and Modulates Circadian Clock Dynamics.
P2860
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P2860
Mutant K-Ras increases GSK-3β gene expression via an ETS-p300 transcriptional complex in pancreatic cancer.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Mutant K-Ras increases GSK-3β ...... complex in pancreatic cancer.
@en
type
label
Mutant K-Ras increases GSK-3β ...... complex in pancreatic cancer.
@en
prefLabel
Mutant K-Ras increases GSK-3β ...... complex in pancreatic cancer.
@en
P2093
P2860
P356
P1433
P1476
Mutant K-Ras increases GSK-3β ...... complex in pancreatic cancer.
@en
P2093
A Harrison
A V Ugolkov
D D Billadeau
M E Fernandez-Zapico
P2860
P2888
P304
P356
10.1038/ONC.2011.90
P407
P577
2011-03-28T00:00:00Z
P5875
P6179
1032715038