The transforming growth factor-beta-inducible matrix protein (beta)ig-h3 interacts with fibronectin.
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Corneal dystrophy-associated R124H mutation disrupts TGFBI interaction with Periostin and causes mislocalization to the lysosomeHigh-level inducible Smad4-reexpression in the cervical cancer cell line C4-II is associated with a gene expression profile that predicts a preferential role of Smad4 in extracellular matrix compositionSolution structure of the Mycobacterium tuberculosis complex protein MPB70: from tuberculosis pathogenesis to inherited human corneal deseaseModulation of Anopheles stephensi gene expression by nitroquine, an antimalarial drug against Plasmodium yoelii infection in the mosquitoLacritin and other autophagy associated proteins in ocular surface health.Fibronectin-guided migration of carcinoma collectivesDifferential expression and processing of transforming growth factor beta induced protein (TGFBIp) in the normal human cornea during postnatal development and agingDefining elastic fiber interactions by molecular fishing: an affinity purification and mass spectrometry approach.Transforming growth factor-β-induced protein (TGFBI) suppresses mesothelioma progression through the Akt/mTOR pathwayHNF1α inhibition triggers epithelial-mesenchymal transition in human liver cancer cell lines.The insoluble TGFBIp fraction of the cornea is covalently linked via a disulfide bond to type XII collagenROCK Inhibition Extends Passage of Pluripotent Stem Cell-Derived Retinal Pigmented Epithelium.Fuchs endothelial corneal dystrophyAssociation of TCF4 and CLU polymorphisms with Fuchs' endothelial dystrophy and implication of CLU and TGFBI proteins in the disease process.TGFBI expression reduces in vitro and in vivo metastatic potential of lung and breast tumor cells.An Arg124His mutation in TGFBI associated to Avellino corneal dystrophy in a Chinese pedigree.The role of novel and known extracellular matrix and adhesion molecules in the homeostatic and regenerative bone marrow microenvironmentTransforming growth Factor-Beta-Induced Protein (TGFBI)/(βig-H3): a matrix protein with dual functions in ovarian cancer.ß3 integrin modulates transforming growth factor beta induced (TGFBI) function and paclitaxel response in ovarian cancer cells.The extracellular matrix protein TGFBI induces microtubule stabilization and sensitizes ovarian cancers to paclitaxelRelease of TGFβig-h3 by gastric myofibroblasts slows tumor growth and is decreased with cancer progression.Two novel VHL targets, TGFBI (BIGH3) and its transactivator KLF10, are up-regulated in renal clear cell carcinoma and other tumors.Microarray analysis of choroid/RPE gene expression in marmoset eyes undergoing changes in ocular growth and refraction.Comparative proteomic analysis of normal and collagen IX null mouse cartilage reveals altered extracellular matrix composition and novel components of the collagen IX interactome.Established and newly proposed mechanisms of chronic cyclosporine nephropathyDecreased catalase expression and increased susceptibility to oxidative stress in primary cultured corneal fibroblasts from patients with granular corneal dystrophy type II.Colocalization of increased transforming growth factor-beta-induced protein (TGFBIp) and Clusterin in Fuchs endothelial corneal dystrophy.Inhibition of human scleral fibroblast cell attachment to collagen type I by TGFBIpEvidence against a blood derived origin for transforming growth factor beta induced protein in corneal disorders caused by mutations in the TGFBI geneBigh3 silencing increases retinoblastoma tumor growth in the murine SV40-TAg-Rb model.Transforming growth factor-beta-induced protein secreted by peritoneal cells increases the metastatic potential of ovarian cancer cells.C-terminal fragment of transforming growth factor beta-induced protein (TGFBIp) is required for apoptosis in human osteosarcoma cells.Initial Suppression of Transforming Growth Factor-β Signaling and Loss of TGFBI Causes Early Alveolar Structural Defects Resulting in Bronchopulmonary Dysplasia.A Case of Transforming Growth Factor-β-Induced Gene-Related Oculorenal Syndrome: Granular Corneal Dystrophy Type II with a Unique Nephropathy.BIGH3 modulates adhesion and migration of hematopoietic stem and progenitor cells.Crystallization and preliminary crystallographic analysis of the fourth FAS1 domain of human BigH3.βig-h3 potentiates the profibrogenic effect of TGFβ signaling on connective tissue progenitor cells through the negative regulation of master chondrogenic genes.TGFBI and CHST6 gene analysis in Chinese stromal corneal dystrophies.Beta ig-h3 induces keratinocyte differentiation via modulation of involucrin and transglutaminase expression through the integrin alpha3beta1 and the phosphatidylinositol 3-kinase/Akt signaling pathway.TGF-β induces oncofetal fibronectin that, in turn, modulates TGF-β superfamily signaling in endothelial cells.
P2860
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P2860
The transforming growth factor-beta-inducible matrix protein (beta)ig-h3 interacts with fibronectin.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
The transforming growth factor ...... h3 interacts with fibronectin.
@en
type
label
The transforming growth factor ...... h3 interacts with fibronectin.
@en
prefLabel
The transforming growth factor ...... h3 interacts with fibronectin.
@en
P2093
P2860
P356
P1476
The transforming growth factor ...... h3 interacts with fibronectin.
@en
P2093
Arthur J Cohen
Beatrice N Engelsberg
Christopher S Adams
J Charles Whitbeck
Joel Rosenbloom
Pamela S Howard
Paul C Billings
William R Abrams
P2860
P304
28003-28009
P356
10.1074/JBC.M106837200
P407
P577
2002-05-28T00:00:00Z