Cytotoxic necrotizing factor-1 contributes to Escherichia coli K1 invasion of the central nervous system.
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Lu/BCAM adhesion glycoprotein is a receptor for Escherichia coli Cytotoxic Necrotizing Factor 1 (CNF1)Defense at the border: the blood-brain barrier versus bacterial foreignersPathogens penetrating the central nervous system: infection pathways and the cellular and molecular mechanisms of invasionSphingosine 1-Phosphate Activation of EGFR As a Novel Target for Meningitic Escherichia coli Penetration of the Blood-Brain BarrierClinical microbiology of bacterial and fungal sepsis in very-low-birth-weight infants.37-kDa laminin receptor precursor modulates cytotoxic necrotizing factor 1-mediated RhoA activation and bacterial uptake.Cloning and expression of the Escherichia coli K1 outer membrane protein A receptor, a gp96 homologue.Use of zebrafish to probe the divergent virulence potentials and toxin requirements of extraintestinal pathogenic Escherichia coli.Cytotoxic Necrotizing Factor-1 (CNF1) does not promote E. coli infection in a murine model of ascending pyelonephritisUse of diagnostic microarrays for determination of virulence gene patterns of Escherichia coli K1, a major cause of neonatal meningitisProteomic analysis of human umbilical vein endothelial cells incubated with Cryptococcus neoformans var. neoformans.Prevention of Escherichia coli K1 penetration of the blood-brain barrier by counteracting the host cell receptor and signaling molecule involved in E. coli invasion of human brain microvascular endothelial cells.YgfZ contributes to secretion of cytotoxic necrotizing factor 1 into outer-membrane vesicles in Escherichia coliArachidonic acid metabolism regulates Escherichia coli penetration of the blood-brain barrierEffects of ompA deletion on expression of type 1 fimbriae in Escherichia coli K1 strain RS218 and on the association of E. coli with human brain microvascular endothelial cells.Emerging molecular targets in the treatment of bacterial meningitis.Hcp family proteins secreted via the type VI secretion system coordinately regulate Escherichia coli K1 interaction with human brain microvascular endothelial cells.Characterizing the pathotype of neonatal meningitis causing Escherichia coli (NMEC).Covert operations of uropathogenic Escherichia coli within the urinary tract.IbeA and OmpA of Escherichia coli K1 exploit Rac1 activation for invasion of human brain microvascular endothelial cellsCryptococcus neoformans activates RhoGTPase proteins followed by protein kinase C, focal adhesion kinase, and ezrin to promote traversal across the blood-brain barrier.The hek outer membrane protein of Escherichia coli strain RS218 binds to proteoglycan and utilizes a single extracellular loop for adherence, invasion, and autoaggregation.gp96 expression in neutrophils is critical for the onset of Escherichia coli K1 (RS218) meningitisConcepts and mechanisms: crossing host barriers.Human Meningitis-Associated Escherichia coli.Mechanisms of microbial traversal of the blood-brain barrier.Complete Genome Sequence of the Escherichia coli PMV-1 Strain, a Model Extraintestinal Pathogenic E. coli Strain Used for Host-Pathogen Interaction Studies.The effects of cytotoxic necrotizing factor 1 expression in the uptake of Escherichia coli K1 by macrophages and the onset of meningitis in newborn mice.Cytotoxic necrotizing factor 1 contributes to Escherichia coli meningitis.Pathogen translocation across the blood-brain barrier.Next generation of non-mammalian blood-brain barrier models to study parasitic infections of the central nervous systemCytotoxic Necrotizing Factors (CNFs)-A Growing Toxin Family.Escherichia coli K1 utilises host macropinocytic pathways for invasion of brain microvascular endothelial cells.mRNA context dependent regulation of cytotoxic necrotizing factor 1 translation by GidA, a tRNA modification enzyme in Escherichia coli.Cleavage of Escherichia coli cytotoxic necrotizing factor 1 is required for full biologic activity.Activation and proteasomal degradation of rho GTPases by cytotoxic necrotizing factor-1 elicit a controlled inflammatory response.The K1 capsule modulates trafficking of E. coli-containing vacuoles and enhances intracellular bacterial survival in human brain microvascular endothelial cells.Pertussis Toxin Exploits Specific Host Cell Signaling Pathways for Promoting Invasion and Translocation of Escherichia coli K1 RS218 in Human Brain-derived Microvascular Endothelial Cells.Escherichia coli K1 meningitis: Analysis of the effects of CNF1 toxin in newborn mice questions its virulence function.Interactions of neuropathogenic Escherichia coli K1 (RS218) and its derivatives lacking genomic islands with phagocytic Acanthamoeba castellanii and nonphagocytic brain endothelial cells.
P2860
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P2860
Cytotoxic necrotizing factor-1 contributes to Escherichia coli K1 invasion of the central nervous system.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Cytotoxic necrotizing factor-1 ...... of the central nervous system.
@en
type
label
Cytotoxic necrotizing factor-1 ...... of the central nervous system.
@en
prefLabel
Cytotoxic necrotizing factor-1 ...... of the central nervous system.
@en
P2093
P2860
P356
P1476
Cytotoxic necrotizing factor-1 ...... of the central nervous system.
@en
P2093
Carol Ann Wass
Jin Woong Chung
Kee Jun Kim
Kwang Sik Kim
Naveed Ahmed Khan
P2860
P304
15607-15612
P356
10.1074/JBC.M112224200
P407
P577
2002-02-27T00:00:00Z