An internal segment (residues 58-119) of the hepatitis B virus X protein is sufficient to activate MAP kinase pathways in mouse liver.
about
Role of a novel functional variant in the PPP2R1A promoter on the regulation of PP2A-Aalpha and the risk of hepatocellular carcinomaHBx-dependent cell cycle deregulation involves interaction with cyclin E/A-cdk2 complex and destabilization of p27Kip1.The mitogenic function of hepatitis B virus X protein resides within amino acids 51 to 140 and is modulated by N- and C-terminal regulatory regions.Hepatitis B virus X protein enhances activation of nuclear factor κB through interaction with valosin-containing protein.The carboxy-terminus of the hepatitis B virus X protein is necessary and sufficient for the activation of hypoxia-inducible factor-1alpha.Specific inhibition of gene expression and transactivation functions of hepatitis B virus X protein and c-myc by small interfering RNAs.Hepatitis B Virus-Associated Hepatocellular Carcinoma and Hepatic Cancer Stem Cells.Molecular Targets in Hepatocarcinogenesis and Implications for Therapy
P2860
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P2860
An internal segment (residues 58-119) of the hepatitis B virus X protein is sufficient to activate MAP kinase pathways in mouse liver.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
An internal segment (residues ...... inase pathways in mouse liver.
@en
type
label
An internal segment (residues ...... inase pathways in mouse liver.
@en
prefLabel
An internal segment (residues ...... inase pathways in mouse liver.
@en
P2093
P2860
P1433
P1476
An internal segment (residues ...... inase pathways in mouse liver.
@en
P2093
P2860
P356
10.1016/S0014-5793(01)02773-9
P407
P577
2001-08-01T00:00:00Z