Genetic abnormalities involved in t(12;21) TEL-AML1 acute lymphoblastic leukemia: analysis by means of array-based comparative genomic hybridization.
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Germinal and Somatic Trisomy 21 Mosaicism: How Common is it, What are the Implications for Individual Carriers and How Does it Come About?Tumor suppressors BTG1 and IKZF1 cooperate during mouse leukemia development and increase relapse risk in B-cell precursor acute lymphoblastic leukemia patients.A new method to detect loss of heterozygosity using cohort heterozygosity comparisons.Pathogenesis of ETV6/RUNX1-positive childhood acute lymphoblastic leukemia and mechanisms underlying its relapseThe origin and nature of tightly clustered BTG1 deletions in precursor B-cell acute lymphoblastic leukemia support a model of multiclonal evolution.The correlation pattern of acquired copy number changes in 164 ETV6/RUNX1-positive childhood acute lymphoblastic leukemiasEbf1 or Pax5 haploinsufficiency synergizes with STAT5 activation to initiate acute lymphoblastic leukemia.Gene expression profiling identifies IRF4-associated molecular signatures in hematological malignanciesCopy number genome alterations are associated with treatment response and outcome in relapsed childhood ETV6/RUNX1-positive acute lymphoblastic leukemia.PU.1 is a major transcriptional activator of the tumour suppressor gene LIMD1.Modeling the evolution of ETV6-RUNX1-induced B-cell precursor acute lymphoblastic leukemia in mice.The serotonin receptor-antagonist ondansetron induces significant increases in the expression of interferon-gamma which correlate with antiproliferative properties in the acute lymphoblastic leukaemia cell line REH.Histone deacetylase inhibition improves differentiation of dendritic cells from leukemic blasts of patients with TEL/AML1-positive acute lymphoblastic leukemia.Abnormalities of the der(12)t(12;21) in ETV6-RUNX1 acute lymphoblastic leukemia.Molecular processes involved in B cell acute lymphoblastic leukaemia.TEL (ETV6)-AML1 (RUNX1) initiates self-renewing fetal pro-B cells in association with a transcriptional program shared with embryonic stem cells in mice.BTG1 deletions do not predict outcome in Down syndrome acute lymphoblastic leukemiaInterphase fluorescent in situ hybridization deletion analysis of the 9p21 region and prognosis in childhood acute lymphoblastic leukaemia (ALL): results from a prospective analysis of 519 Nordic patients treated according to the NOPHO-ALL 2000 proto
P2860
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P2860
Genetic abnormalities involved in t(12;21) TEL-AML1 acute lymphoblastic leukemia: analysis by means of array-based comparative genomic hybridization.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Genetic abnormalities involved ...... arative genomic hybridization.
@en
type
label
Genetic abnormalities involved ...... arative genomic hybridization.
@en
prefLabel
Genetic abnormalities involved ...... arative genomic hybridization.
@en
P2093
P2860
P1433
P1476
Genetic abnormalities involved ...... arative genomic hybridization.
@en
P2093
Kanji Sugita
Keizo Horibe
Kimikazu Matsumoto
Kumiko Goi
Masao Seto
Ryuzo Ueda
Shinobu Tsuzuki
Shinpei Nakazawa
Sivasundaram Karnan
P2860
P304
P356
10.1111/J.1349-7006.2007.00443.X
P577
2007-03-21T00:00:00Z