N-glycosylation affects the molecular organization and stability of E-cadherin junctions.
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Overexpression of DPAGT1 leads to aberrant N-glycosylation of E-cadherin and cellular discohesion in oral cancerHijacking the Hexosamine Biosynthetic Pathway to Promote EMT-Mediated Neoplastic PhenotypesProtein N-glycosylation in oral cancer: dysregulated cellular networks among DPAGT1, E-cadherin adhesion and canonical Wnt signalingThe Integrated Role of Wnt/β-Catenin, N-Glycosylation, and E-Cadherin-Mediated Adhesion in Network DynamicsE-cadherin roles in animal biology: A perspective on thyroid hormone-influenceMembrane biophysics define neuron and astrocyte progenitors in the neural lineageChallenging the roles of CD44 and lipolysis stimulated lipoprotein receptor in conveying Clostridium perfringens iota toxin cytotoxicity in breast cancer.N-glycosylation status of E-cadherin controls cytoskeletal dynamics through the organization of distinct β-catenin- and γ-catenin-containing AJs.Hypoglycosylated E-cadherin promotes the assembly of tight junctions through the recruitment of PP2A to adherens junctions.N-glycosylation gene DPAGT1 is a target of the Wnt/beta-catenin signaling pathway.N-glycosylation regulates ADAM8 processing and activationGlycan structures contain information for the spatial arrangement of glycoproteins in the plasma membrane.Epithelial junctions depend on intercellular trans-interactions between the Na,K-ATPase β₁ subunits.Pancreatic cancer cell glycosylation regulates cell adhesion and invasion through the modulation of α2β1 integrin and E-cadherin function.Roles of N-acetylglucosaminyltransferase III in epithelial-to-mesenchymal transition induced by transforming growth factor β1 (TGF-β1) in epithelial cell lines.The Na-K-ATPase α₁β₁ heterodimer as a cell adhesion molecule in epithelia.Aberrant amplification of the crosstalk between canonical Wnt signaling and N-glycosylation gene DPAGT1 promotes oral cancer.Galectin-3 protein regulates mobility of N-cadherin and GM1 ganglioside at cell-cell junctions of mammary carcinoma cellsSecreted Hsp90 is a novel regulator of the epithelial to mesenchymal transition (EMT) in prostate cancer.Coordinate regulation of N-glycosylation gene DPAGT1, canonical Wnt signaling and E-cadherin adhesion.Preventing E-cadherin aberrant N-glycosylation at Asn-554 improves its critical function in gastric cancer.N-glycosylation induces the CTHRC1 protein and drives oral cancer cell migration.Identification of candidate biomarkers with cancer-specific glycosylation in the tissue and serum of endometrioid ovarian cancer patients by glycoproteomic analysis.Regulation of homotypic cell-cell adhesion by branched N-glycosylation of N-cadherin extracellular EC2 and EC3 domains.Modulation of N-glycosylation by mesalamine facilitates membranous E-cadherin expression in colon epithelial cells.Potential roles of N-glycosylation in cell adhesion.E-cadherin dysfunction in gastric cancer--cellular consequences, clinical applications and open questions.Nesting of colon and ovarian cancer cells in the endothelial niche is associated with alterations in glycan and lipid metabolism.Predominant Expression of Hybrid N-Glycans Has Distinct Cellular Roles Relative to Complex and Oligomannose N-Glycans.Inverse correlation between the extent of N-glycan branching and intercellular adhesion in epithelia. Contribution of the Na,K-ATPase beta1 subunit.Adherens Junctions on the Move-Membrane Trafficking of E-Cadherin.Dystroglycan function is a novel determinant of tumor growth and behavior in prostate cancer.The role of N-acetylglucosaminyltransferase III and V in the post-transcriptional modifications of E-cadherin.Core fucosylation of E-cadherin enhances cell-cell adhesion in human colon carcinoma WiDr cells.Lack of phosphomannomutase 2 affects Xenopus laevis morphogenesis and the non-canonical Wnt5a/Ror2 signalling.O-mannosylation and N-glycosylation: two coordinated mechanisms regulating the tumour suppressor functions of E-cadherin in cancer.Label-free mass spectrometry proteome quantification of human embryonic kidney cells following 24 hours of sialic acid overproductionWnt/beta-catenin signaling down-regulates N-acetylglucosaminyltransferase III expression: the implications of two mutually exclusive pathways for regulation.Multiple post-translational modifications regulate E-cadherin transport during apoptosis.Prebiotic Oligosaccharides Potentiate Host Protective Responses against L. Monocytogenes Infection.
P2860
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P2860
N-glycosylation affects the molecular organization and stability of E-cadherin junctions.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
N-glycosylation affects the molecular organization and stability of E-cadherin junctions.
@en
type
label
N-glycosylation affects the molecular organization and stability of E-cadherin junctions.
@en
prefLabel
N-glycosylation affects the molecular organization and stability of E-cadherin junctions.
@en
P2860
P356
P1476
N-glycosylation affects the molecular organization and stability of E-cadherin junctions.
@en
P2093
Aneta Liwosz
Tianlei Lei
P2860
P304
23138-23149
P356
10.1074/JBC.M512621200
P407
P577
2006-05-08T00:00:00Z