Transcriptional control of human papillomavirus (HPV) oncogene expression: composition of the HPV type 18 upstream regulatory region.
about
Distinct modes of regulation of transcription of hepatitis B virus by the nuclear receptors HNF4alpha and COUP-TF1Heparin (GAG-hed) inhibits LCR activity of human papillomavirus type 18 by decreasing AP1 binding.Induction of the human papillomavirus type 31 late promoter requires differentiation but not DNA amplificationVariant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Novel binding sites for regulatory factors in the human papillomavirus type 18 enhancer and promoter identified by in vivo footprintingCritical roles of nuclear receptor response elements in replication of hepatitis B virus.Cervical cancer stem cells manifest radioresistance: Association with upregulated AP-1 activity.Two Sp1/Sp3 binding sites in the major immediate-early proximal enhancer of human cytomegalovirus have a significant role in viral replication.Anticancer property of Bryophyllum pinnata (Lam.) Oken. leaf on human cervical cancer cellsThe upstream regulatory region of human papillomavirus type 31 is insensitive to glucocorticoid induction.Pathogenesis of human papillomaviruses in differentiating epithelia.Control of HPV 18 DNA replication by cellular and viral transcription factors.Human papillomavirus type 31 replication modes during the early phases of the viral life cycle depend on transcriptional and posttranscriptional regulation of E1 and E2 expression.Berberine modulates AP-1 activity to suppress HPV transcription and downstream signaling to induce growth arrest and apoptosis in cervical cancer cells.Curcumin modulates cellular AP-1, NF-kB, and HPV16 E6 proteins in oral cancer.The effects of DNA methylation and histone deacetylase inhibitors on human papillomavirus early gene expression in cervical cancer, an in vitro and clinical studyPromoters for synthesis of the pre-C and pregenomic mRNAs of human hepatitis B virus are genetically distinct and differentially regulatedPapillomavirus-Associated Tumor Formation Critically Depends on c-Fos Expression Induced by Viral Protein E2 and Bromodomain Protein Brd4.Transcription activities of human papillomavirus type 11 E6 promoter-proximal elements in raft and submerged cultures of foreskin keratinocytes.Post-Transcriptional Regulation of KLF4 by High-Risk Human Papillomaviruses Is Necessary for the Differentiation-Dependent Viral Life Cycle.Recent advances in the search for antiviral agents against human papillomaviruses.Selective participation of c-Jun with Fra-2/c-Fos promotes aggressive tumor phenotypes and poor prognosis in tongue cancer.Nucleolin as activator of human papillomavirus type 18 oncogene transcription in cervical cancerDifferential regulation of the pre-C and pregenomic promoters of human hepatitis B virus by members of the nuclear receptor superfamilyThe E2 transcriptional repressor can compensate for Sp1 activation of the human papillomavirus type 18 early promoter.NFI-Ski interactions mediate transforming growth factor beta modulation of human papillomavirus type 16 early gene expression.Differentiation-dependent changes in levels of C/EBPβ repressors and activators regulate human papillomavirus type 31 late gene expression.HMG-I(Y) and the CBP/p300 coactivator are essential for human papillomavirus type 18 enhanceosome transcriptional activity.Mutational analysis of adeno-associated virus Rep protein-mediated inhibition of heterologous and homologous promotersRegulation of human papillomavirus transcription by the differentiation-dependent epithelial factor Epoc-1/skn-1a.Antioxidant-induced changes of the AP-1 transcription complex are paralleled by a selective suppression of human papillomavirus transcription.Induction of the upstream regulatory region of human papillomavirus type 31 by dexamethasone is differentiation dependent.Different mechanisms contribute to the E2-mediated transcriptional repression of human papillomavirus type 18 viral oncogenes.Interference with energy metabolism by 5-aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside induces HPV suppression in cervical carcinoma cells and apoptosis in the absence of LKB1.NF-Y loss triggers p53 stabilization and apoptosis in HPV18-positive cells by affecting E6 transcription.An enhanceosome containing the Jun B/Fra-2 heterodimer and the HMG-I(Y) architectural protein controls HPV 18 transcription.Oxidative stress in female cancers.
P2860
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P2860
Transcriptional control of human papillomavirus (HPV) oncogene expression: composition of the HPV type 18 upstream regulatory region.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Transcriptional control of hum ...... 18 upstream regulatory region.
@en
type
label
Transcriptional control of hum ...... 18 upstream regulatory region.
@en
prefLabel
Transcriptional control of hum ...... 18 upstream regulatory region.
@en
P2860
P1433
P1476
Transcriptional control of hum ...... 18 upstream regulatory region
@en
P2093
Hoppe-Seyler F
P2860
P304
P407
P577
1993-11-01T00:00:00Z