Free fatty acids repress the GLUT4 gene expression in cardiac muscle via novel response elements. - Wikidata - wikimedia - TriplyDB

Free fatty acids repress the GLUT4 gene expression in cardiac muscle via novel response elements.

Free fatty acids repress the GLUT4 gene expression in cardiac muscle via novel response elements.

http://www.wikidata.org/entity/Q38322283

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PPARs and the cardiovascular system Effects of PPARalpha on cardiac glucose metabolism: a transcriptional equivalent of the glucose-fatty acid cycle?Hypertriglyceridemia: a too long unfairly neglected major cardiovascular risk factor.Obesity and breast cancer: the roles of peroxisome proliferator-activated receptor-γ and plasminogen activator inhibitor-1 Role of adiponectin and free fatty acids on the association between abdominal visceral fat and insulin resistance.Central obesity, type 2 diabetes and insulin: exploring a pathway full of thorns.Metabolic diseases and pro- and prebiotics: Mechanistic insights Enhanced Nrf2 activity worsens insulin resistance, impairs lipid accumulation in adipose tissue, and increases hepatic steatosis in leptin-deficient mice.Alterations in energy metabolism in cardiomyopathies.Obesity and free fatty acids.Transcriptional regulation of the insulin-responsive glucose transporter GLUT4 gene: from physiology to pathology.Taking diabetes to heart--deregulation of myocardial lipid metabolism in diabetic cardiomyopathy.Suppression of the GLUT4 adaptive response to exercise in fructose-fed rats.My Sweetheart Is Broken: Role of Glucose in Diabetic Cardiomyopathy.Review article: Left ventricular dysfunction and heart failure in metabolic syndrome and diabetes without overt coronary artery disease--do we need to screen our patients?Diabetic Cardiomyopathy: An Immunometabolic Perspective.Fatty acid binding receptors in intestinal physiology and pathophysiology Metabolic abnormalities of the heart in type II diabetes.Inhibition of protein kinase B by palmitate in the insulin signaling of HepG2 cells and the preventive effect of arachidonic acid on insulin resistance.Methylene blue decreases mitochondrial lysine acetylation in the diabetic heart Effects of hormone-sensitive lipase disruption on cardiac energy metabolism in response to fasting and refeeding.Western diet impairs metabolic remodelling and contractile efficiency in cardiac hypertrophy.Exposure of Pregnant Mice to Triclosan Causes Insulin Resistance via Thyroxine Reduction.Effects of crocin and voluntary exercise, alone or combined, on heart VEGF-A and HOMA-IR of HFD/STZ induced type 2 diabetic rats.Effect of Propofol Continuous-Rate Infusion on Intravenous Glucose Tolerance Test in Dogs.

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P2860

Free fatty acids repress the GLUT4 gene expression in cardiac muscle via novel response elements.

http://www.wikidata.org/entity/Q38322283

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

Free fatty acids repress the G ...... e via novel response elements.

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type

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Free fatty acids repress the G ...... e via novel response elements.

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Free fatty acids repress the G ...... e via novel response elements.

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P1433

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P1433

Journal of Biological Chemistry

P1476

Free fatty acids repress the G ...... e via novel response elements.

@en

P2093

Chava Harel

http://www.w3.org/2001/XMLSchema#string

Eddy Karnieli

http://www.w3.org/2001/XMLSchema#string

Fabiana Bar-Yoseph

http://www.w3.org/2001/XMLSchema#string

Michal Armoni

http://www.w3.org/2001/XMLSchema#string

Simcha Milo

http://www.w3.org/2001/XMLSchema#string

P2860

Cardiac hypertrophy with preserved contractile function after selective deletion of GLUT4 from the heart

Reversal of obesity- and diet-induced insulin resistance with salicylates or targeted disruption of Ikkbeta

The organization, promoter analysis, and expression of the human PPARgamma gene

Corepressors selectively control the transcriptional activity of PPARgamma in adipocytes

mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer

Intensive insulin therapy in critically ill patients

Arachidonic acid stimulates glucose uptake in 3T3-L1 adipocytes by increasing GLUT1 and GLUT4 levels at the plasma membrane. Evidence for involvement of lipoxygenase metabolites and peroxisome proliferator-activated receptor gamma.

Free fatty acids, insulin resistance, and type 2 diabetes mellitus.

Regulation of gene expression by fatty acids.

Inhibition of cellular proliferation through IkappaB kinase-independent and peroxisome proliferator-activated receptor gamma-dependent repression of cyclin D1.

P304

34786-34795

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scholarly article

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10.1074/JBC.M502740200

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41

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280

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2005-08-10T00:00:00Z

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7664720

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16096283

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