Low doses of bisphenol A induce gene expression related to lipid synthesis and trigger triglyceride accumulation in adult mouse liver.
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Non-monotonic dose-response relationships and endocrine disruptors: a qualitative method of assessmentExposure to bisphenol-A during pregnancy partially mimics the effects of a high-fat diet altering glucose homeostasis and gene expression in adult male miceEDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting ChemicalsSex differences in the association of urinary bisphenol-A concentration with selected indices of glucose homeostasis among U.S. adultsIdentification of modulators of the nuclear receptor peroxisome proliferator-activated receptor α (PPARα) in a mouse liver gene expression compendiumLiver transcriptome analysis of Atlantic cod (Gadus morhua) exposed to PCB 153 indicates effects on cell cycle regulation and lipid metabolismNuclear receptors and endocrine disruptors in fetal and neonatal testes: a gapped landscape.Endocrine disrupting chemicals in mixture and obesity, diabetes and related metabolic disorders.Evaluation of low doses BPA-induced perturbation of glycemia by toxicogenomics points to a primary role of pancreatic islets and to the mechanism of toxicity.Toxic effects of maternal zearalenone exposure on uterine capacity and fetal development in gestation rats.Non-monotonic dose responses in studies of endocrine disrupting chemicals: bisphenol a as a case studyBisphenol A, obesity, and type 2 diabetes mellitus: genuine concern or unnecessary preoccupation?Environmental endocrine disruption of energy metabolism and cardiovascular risk.Bisphenol A in polycystic ovary syndrome and its association with liver-spleen axis.Bisphenol A exposure enhances atherosclerosis in WHHL rabbits.The g0/g1 switch gene 2 is an important regulator of hepatic triglyceride metabolism.Early-life exposure to bisphenol a induces liver injury in rats involvement of mitochondria-mediated apoptosisMetabolic outcome of female mice exposed to a mixture of low-dose pollutants in a diet-induced obesity model.Developmental bisphenol A (BPA) exposure leads to sex-specific modification of hepatic gene expression and epigenome at birth that may exacerbate high-fat diet-induced hepatic steatosis.Environmentally Relevant Dose of Bisphenol A Does Not Affect Lipid Metabolism and Has No Synergetic or Antagonistic Effects on Genistein's Beneficial Roles on Lipid MetabolismBisphenol A and peripheral arterial disease: results from the NHANES.Bisphenol A Exposure May Induce Hepatic Lipid Accumulation via Reprogramming the DNA Methylation Patterns of Genes Involved in Lipid Metabolism.Keap1 knockdown increases markers of metabolic syndrome after long-term high fat diet feedingAdipocytes under assault: environmental disruption of adipose physiology.Bisphenol A promotes cholesterol absorption in Caco-2 cells by up-regulation of NPC1L1 expression.Chronic exposure to bisphenol A can accelerate atherosclerosis in high-fat-fed apolipoprotein E knockout mice.Is bisphenol A an environmental obesogen?Bisphenol A: Targeting metabolic tissues.Bisphenol a induces steatosis in HepaRG cells using a model of perinatal exposure.Molecular targets of developmental exposure to bisphenol A in diabesity: a focus on endoderm-derived organs.Maternal bisphenol A exposure alters rat offspring hepatic and skeletal muscle insulin signaling protein abundance.Endocrine-disrupting chemicals and the regulation of energy balance.Effects of Low-Dose Developmental Bisphenol A Exposure on Metabolic Parameters and Gene Expression in Male and Female Fischer 344 Rat Offspring.Bisphenol A exposure induces metabolic disorders and enhances atherosclerosis in hyperlipidemic rabbits.Sex differences, endogenous sex-hormone hormones, sex-hormone binding globulin, and exogenous disruptors in diabetes and related metabolic outcomes.Dietary oleic acid regulates hepatic lipogenesis through a liver X receptor-dependent signalingLow-Dose Bisphenol A Increases Bile Duct Proliferation in Juvenile Rats: A Possible Evidence for Risk of Liver Cancer in the Exposed Population?Studies on the potential protective effect of cinnamon against bisphenol A- and octylphenol-induced oxidative stress in male albino rats.Identification of mechanisms of action of bisphenol a-induced human preadipocyte differentiation by transcriptional profiling.Hepatic and renal functions in growing male rats after bisphenol A and octylphenol exposure.
P2860
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P2860
Low doses of bisphenol A induce gene expression related to lipid synthesis and trigger triglyceride accumulation in adult mouse liver.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Low doses of bisphenol A induc ...... mulation in adult mouse liver.
@en
type
label
Low doses of bisphenol A induc ...... mulation in adult mouse liver.
@en
prefLabel
Low doses of bisphenol A induc ...... mulation in adult mouse liver.
@en
P2093
P50
P356
P1433
P1476
Low doses of bisphenol A induc ...... umulation in adult mouse liver
@en
P2093
Alain Paris
Alice Marmugi
Arnaud Polizzi
Frédéric Lasserre
Justine Bertrand-Michel
Laïla Mselli-Lakhal
Thierry Pineau
P304
P356
10.1002/HEP.24685
P407
P577
2011-12-22T00:00:00Z