Nuclear integration of glucocorticoid receptor and nuclear factor-kappaB signaling by CREB-binding protein and steroid receptor coactivator-1.
about
Receptor interacting protein RIP140 inhibits both positive and negative gene regulation by glucocorticoidsModulation of NF-kappaB-dependent transcription and cell survival by the SIRT1 deacetylase.Glucocorticoid receptor inhibits transforming growth factor-beta signaling by directly targeting the transcriptional activation function of Smad3Modulation of retinoid signaling by a cytoplasmic viral protein via sequestration of Sp110b, a potent transcriptional corepressor of retinoic acid receptor, from the nucleusTherapeutic potential of inhibition of the NF-kappaB pathway in the treatment of inflammation and cancerGlucocorticoids: do we know how they work?Rationale for Prolonged Glucocorticoid Use in Pediatric ARDS: What the Adults Can Teach UsAcetylation of RelA at discrete sites regulates distinct nuclear functions of NF-kappaB.JAB1 interacts with both the progesterone receptor and SRC-1Identification of glucocorticoid receptor domains involved in transrepression of transforming growth factor-beta actionTranscriptional activation by STAT6 requires the direct interaction with NCoA-1The POU domain factor Skin-1a represses the keratin 14 promoter independent of DNA binding. A possible role for interactions between Skn-1a and CREB-binding protein/p300Functional interaction of STAT3 transcription factor with the coactivator NcoA/SRC1aNuclear factor kappa B is involved in lipopolysaccharide-stimulated induction of interferon regulatory factor-1 and GAS/GAF DNA-binding in human umbilical vein endothelial cellsExactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptorSIRT1 activators suppress inflammatory responses through promotion of p65 deacetylation and inhibition of NF-κB activityIneffectiveness of histone deacetylase inhibitors to induce apoptosis involves the transcriptional activation of NF-kappa B through the Akt pathway.Identification of a negative regulatory cis-element in the enhancer core region of the prostate-specific antigen promoter: implications for intersection of androgen receptor and nuclear factor-kappaB signalling in prostate cancer cells.Modification of RelA by O-linked N-acetylglucosamine links glucose metabolism to NF-κB acetylation and transcription.IkappaB kinase alpha-mediated derepression of SMRT potentiates acetylation of RelA/p65 by p300.NF-kappaB activation by N-CAM and cytokines in astrocytes is regulated by multiple protein kinases and redox modulation.Distinct p300-responsive mechanisms promote caspase-dependent apoptosis by human T-cell lymphotropic virus type 1 Tax protein.Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatasesNuclear receptors: mediators and modifiers of inflammation-induced cholestasis.Glucocorticoid receptor recruitment of histone deacetylase 2 inhibits interleukin-1beta-induced histone H4 acetylation on lysines 8 and 12.Molecular mechanisms of glucocorticoid action: what is important?Transcriptional activation by NF-kappaB requires multiple coactivatorsBiochemical analysis of distinct activation functions in p300 that enhance transcription initiation with chromatin templates.Peroxisome proliferator-activated receptor gamma-dependent repression of the inducible nitric oxide synthase gene.Presentation at the National Asthma Campaign International Congress, June 1999. Controlling the inflammatory response through transcriptional mechanisms.Increased p300 expression inhibits glucocorticoid receptor-T-cell receptor antagonism but does not affect thymocyte positive selectionMicrotubule-dependent subcellular redistribution of the transcriptional coactivator p/CIP.NCX-1015, a nitric-oxide derivative of prednisolone, enhances regulatory T cells in the lamina propria and protects against 2,4,6-trinitrobenzene sulfonic acid-induced colitis in mice.Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.Direct GR Binding Sites Potentiate Clusters of TF Binding across the Human Genome.Ligand-induced differentiation of glucocorticoid receptor (GR) trans-repression and transactivation: preferential targetting of NF-kappaB and lack of I-kappaB involvement.Enhancement of 1 alpha,25-dihydroxyvitamin D(3)-induced differentiation of human leukaemia HL-60 cells into monocytes by parthenolide via inhibition of NF-kappa B activityCytoplasmic catalytic subunit of protein kinase A mediates cross-repression by NF-kappa B and the glucocorticoid receptor.Suppression of PTEN expression by NF-kappa B prevents apoptosis.Pathophysiological factors affecting CAR gene expression.
P2860
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P2860
Nuclear integration of glucocorticoid receptor and nuclear factor-kappaB signaling by CREB-binding protein and steroid receptor coactivator-1.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
Nuclear integration of glucoco ...... teroid receptor coactivator-1.
@en
type
label
Nuclear integration of glucoco ...... teroid receptor coactivator-1.
@en
prefLabel
Nuclear integration of glucoco ...... teroid receptor coactivator-1.
@en
P2093
P2860
P356
P1476
Nuclear integration of glucoco ...... teroid receptor coactivator-1.
@en
P2093
Gerritsen ME
Rosenfeld MG
Sheppard KA
Williams AJ
P2860
P304
29291-29294
P356
10.1074/JBC.273.45.29291
P407
P577
1998-11-01T00:00:00Z