Clostridium difficile toxin A induces expression of the stress-induced early gene product RhoB.
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Clostridium difficile Toxins A and B: Insights into Pathogenic Properties and Extraintestinal EffectsThe Role of Rho GTPases in Toxicity of Clostridium difficile ToxinsRac1-regulated endothelial radiation response stimulates extravasation and metastasis that can be blocked by HMG-CoA reductase inhibitorsHigh temporal resolution of glucosyltransferase dependent and independent effects of Clostridium difficile toxins across multiple cell types.In vivo physiological and transcriptional profiling reveals host responses to Clostridium difficile toxin A and toxin BClostridium difficile toxin B differentially affects GPCR-stimulated Ca2+ responses in macrophages: independent roles for Rho and PLA2.Transcriptional profiling of Clostridium difficile and Caco-2 cells during infection.Yersinia enterocolitica YopT and Clostridium difficile toxin B induce expression of GILZ in epithelial cellsEffector triggered immunityRho-glucosylating Clostridium difficile toxins A and B: new insights into structure and function.Regulation of glioblastoma cell invasion by PKC iota and RhoB.Identification of a novel virulence factor in Clostridium difficile that modulates toxin sensitivity of cultured epithelial cells.MAPK-activated protein kinase 2 contributes to Clostridium difficile-associated inflammationClostridial toxins.Down-regulation of interleukin-16 in human mast cells HMC-1 by Clostridium difficile toxins A and B.Glucosyltransferase-dependent and -independent effects of TcdB on the proteome of HEp-2 cells.Application of mutated Clostridium difficile toxin A for determination of glucosyltransferase-dependent effects.Impact of clostridial glucosylating toxins on the proteome of colonic cells determined by isotope-coded protein labeling and LC-MALDI.Time-resolved cellular effects induced by TcdA from Clostridium difficile.Toxin A of the nosocomial pathogen Clostridium difficile induces primary effects in the proteome of HEp-2 cells.Difference in the cytotoxic effects of toxin B from Clostridium difficile strain VPI 10463 and toxin B from variant Clostridium difficile strain 1470.Suppression of NF-kappaB activation by Entamoeba histolytica in intestinal epithelial cells is mediated by heat shock protein 27.Cholesterol-dependent pore formation of Clostridium difficile toxin A.Secreted Compounds of the Probiotic Bacillus clausii Strain O/C Inhibit the Cytotoxic Effects Induced by Clostridium difficile and Bacillus cereus Toxins.Cellular stability of Rho-GTPases glucosylated by Clostridium difficile toxin B.Autoproteolytic cleavage mediates cytotoxicity of Clostridium difficile toxin A.Cytoprotective effect of the small GTPase RhoB expressed upon treatment of fibroblasts with the Ras-glucosylating Clostridium sordellii lethal toxin.Haemorrhagic toxin and lethal toxin from Clostridium sordellii strain vpi9048: molecular characterization and comparative analysis of substrate specificity of the large clostridial glucosylating toxins.Human neutrophils are activated by a peptide fragment of Clostridium difficile toxin B presumably via formyl peptide receptor.
P2860
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P2860
Clostridium difficile toxin A induces expression of the stress-induced early gene product RhoB.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年學術文章
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name
Clostridium difficile toxin A ...... duced early gene product RhoB.
@en
type
label
Clostridium difficile toxin A ...... duced early gene product RhoB.
@en
prefLabel
Clostridium difficile toxin A ...... duced early gene product RhoB.
@en
P2093
P2860
P356
P1476
Clostridium difficile toxin A ...... duced early gene product RhoB.
@en
P2093
Gerhard Fritz
Harald Genth
Helma Tatge
Jürgen Borlak
Ralf Gerhard
Thomas Thum
P2860
P304
P356
10.1074/JBC.M406014200
P407
P577
2004-11-05T00:00:00Z