Histone deacetylase inhibitors, valproic acid and trichostatin-A induce apoptosis and affect acetylation status of p53 in ERG-positive prostate cancer cells.
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PCA3 and TMPRSS2-ERG gene fusions as diagnostic biomarkers for prostate cancerERG transcriptional networks in primary acute leukemia cells implicate a role for ERG in deregulated kinase signalingAlcohol-induced serotonergic modulation: the role of histone deacetylasesTargeting the DNA-binding activity of the human ERG transcription factor using new heterocyclic dithiophene diamidinesChromatin remodeling, cell proliferation and cell death in valproic acid-treated HeLa cellsOncogenic activation of ERG: A predominant mechanism in prostate cancer.Defining the role of histone deacetylases in the inhibition of mammary carcinogenesis by dietary energy restriction (DER): effects of suberoylanilide hydroxamic acid (SAHA) and DER in a rat model.Autophagy is differentially induced in prostate cancer LNCaP, DU145 and PC-3 cells via distinct splicing profiles of ATG5.A Phase I Protocol of Hydralazine and Valproic Acid in Advanced, Previously Treated Solid Cancers.Histone deacetylases and mechanisms of regulation of gene expressionQuantitative Measurement of Histone Tail Acetylation Reveals Stage-Specific Regulation and Response to Environmental Changes during Drosophila Development.Mechanisms of trichostatin A inhibiting AGS proliferation and identification of lysine-acetylated proteinsValproic acid shows a potent antitumor effect with alteration of DNA methylation in neuroblastomaTrichostatin A Enhances the Apoptotic Potential of Palladium Nanoparticles in Human Cervical Cancer Cells.Androgen receptor and gene network: Micromechanics reassemble the signaling machinery of TMPRSS2-ERG positive prostate cancer cells.Ets Related Gene and Smad3 Proteins Collaborate to Activate Transforming Growth Factor-Beta Mediated Signaling Pathway in ETS Related Gene-Positive Prostate Cancer Cells.Molecular Mechanism of Activation of Transforming Growth Factor Beta/Smads Signaling Pathway in Ets Related Gene-Positive Prostate Cancers.Anti-Epileptic Drug Targets Ewing Sarcoma.Nephrotoxicity of epigenetic inhibitors used for the treatment of cancer.Molecular Mechanism of β-Catenin Signaling Pathway Inactivation in ETV1-Positive Prostate Cancers.Effect of Genistein in Comparison with Trichostatin A on Reactivation of DNMTs Genes in Hepatocellular Carcinoma.
P2860
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P2860
Histone deacetylase inhibitors, valproic acid and trichostatin-A induce apoptosis and affect acetylation status of p53 in ERG-positive prostate cancer cells.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Histone deacetylase inhibitors ...... ositive prostate cancer cells.
@en
type
label
Histone deacetylase inhibitors ...... ositive prostate cancer cells.
@en
prefLabel
Histone deacetylase inhibitors ...... ositive prostate cancer cells.
@en
P2093
P2860
P356
P1476
Histone deacetylase inhibitors ...... ositive prostate cancer cells.
@en
P2093
E Shyam P Reddy
Ganapathy K Bhat
Roland Matthews
Shubhalaxmi Kayarthodi
Veena N Rao
Wendell S Fortson
William E Grizzle
Yasuo Fujimura
P2860
P304
P356
10.3892/IJO.2011.1014
P577
2011-04-21T00:00:00Z