Insulin resistance does not diminish eNOS expression, phosphorylation, or binding to HSP-90. - Wikidata - wikimedia - TriplyDB

Insulin resistance does not diminish eNOS expression, phosphorylation, or binding to HSP-90.

Insulin resistance does not diminish eNOS expression, phosphorylation, or binding to HSP-90.

http://www.wikidata.org/entity/Q38338557

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Arginase promotes endothelial dysfunction and hypertension in obese rats Deletion of protein tyrosine phosphatase 1b improves peripheral insulin resistance and vascular function in obese, leptin-resistant mice via reduced oxidant tone.Variations of brain endothelial nitric oxide synthase concentration in rat and mouse cortex Restoration of coronary endothelial function in obese Zucker rats by a low-carbohydrate diet.Pulmonary arterial dysfunction in insulin resistant obese Zucker rats.Cardiac NO signalling in the metabolic syndrome.Spatial heterogeneity in skeletal muscle microvascular blood flow distribution is increased in the metabolic syndrome Deletion of Protein Tyrosine Phosphatase 1B (PTP1B) Enhances Endothelial Cyclooxygenase 2 Expression and Protects Mice from Type 1 Diabetes-Induced Endothelial Dysfunction Influence of obesity and metabolic dysfunction on the endothelial control in the coronary circulation.Improved endothelial dysfunction by Cynanchum wilfordii in apolipoprotein E(-/-) mice fed a high fat/cholesterol diet.Impact of obesity and insulin resistance on vasomotor tone: nitric oxide and beyond.Obesity and vascular dysfunction.Amyloid β peptide-induced inhibition of endothelial nitric oxide production involves oxidative stress-mediated constitutive eNOS/HSP90 interaction and disruption of agonist-mediated Akt activation.Contribution of insulin and Akt1 signaling to endothelial nitric oxide synthase in the regulation of endothelial function and blood pressure Mechanisms of vascular insulin resistance: a substitute Akt?Impairment of cardiac insulin signaling in fructose-fed ovariectomized female Wistar rats.Cyclooxygenase-2 preserves flow-mediated remodelling in old obese Zucker rat mesenteric arteries.Reduced nitric oxide bioavailability contributes to skeletal muscle microvessel rarefaction in the metabolic syndrome.Oxidant stress and constrictor reactivity impair cerebral artery dilation in obese Zucker rats.Differential structural and functional changes in penile and coronary arteries from obese Zucker rats.Metabolic syndrome increases endogenous carbon monoxide production to promote hypertension and endothelial dysfunction in obese Zucker rats.Nitric oxide bioavailability and not production is first altered during the onset of insulin resistance in sucrose-fed rats.Chronic Remote Ischemic Conditioning Is Cerebroprotective and Induces Vascular Remodeling in a VCID Model.

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P2860

Insulin resistance does not diminish eNOS expression, phosphorylation, or binding to HSP-90.

http://www.wikidata.org/entity/Q38338557

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Insulin resistance does not di ...... ylation, or binding to HSP-90.

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type

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Insulin resistance does not di ...... ylation, or binding to HSP-90.

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Insulin resistance does not di ...... ylation, or binding to HSP-90.

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AJPHEART.00280.2004

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American Journal of Physiology - Heart and Circulatory Physiology

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Insulin resistance does not di ...... ylation, or binding to HSP-90.

@en

P2093

Bruce E Kemp

http://www.w3.org/2001/XMLSchema#string

David Fulton

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David W Stepp

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M Brennan Harris

http://www.w3.org/2001/XMLSchema#string

Mario B Marrero

http://www.w3.org/2001/XMLSchema#string

Richard C Venema

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Insulin-dependent activation of endothelial nitric oxide synthase is impaired by O-linked glycosylation modification of signaling proteins in human coronary endothelial cells

A simplified system for generating recombinant adenoviruses

Pulmonary phosphatidic acid phosphatase and lipid phosphate phosphohydrolase

Molecular cloning of rat leptin receptor isoform complementary DNAs--identification of a missense mutation in Zucker fatty (fa/fa) rats

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

Impaired vagal reflex activity in insulin-resistant rats

Abnormal biopterin metabolism is a major cause of impaired endothelium-dependent relaxation through nitric oxide/O2- imbalance in insulin-resistant rat aorta

Domain mapping studies reveal that the M domain of hsp90 serves as a molecular scaffold to regulate Akt-dependent phosphorylation of endothelial nitric oxide synthase and NO release

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide

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scholarly article

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10.1152/AJPHEART.00280.2004

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6

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2004-07-22T00:00:00Z

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15271670

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phosphorylation