Cardiolipin is not required for Bax-mediated cytochrome c release from yeast mitochondria.
about
The mitochondrial TOM complex is required for tBid/Bax-induced cytochrome c releasePrediction of protein orientation upon immobilization on biological and nonbiological surfacesCardiolipin deficiency leads to decreased cardiolipin peroxidation and increased resistance of cells to apoptosisPermeabilization of the mitochondrial outer membrane by Bax/truncated Bid (tBid) proteins as sensitized by cardiolipin hydroperoxide translocation: mechanistic implications for the intrinsic pathway of oxidative apoptosisCardiolipin remodeling: a regulatory hub for modulating cardiolipin metabolism and functionReactive oxygen species in inflammation and tissue injuryElimination of the unnecessary: intra- and extracellular signaling by anionic phospholipidsCardiolipin is essential for organization of complexes III and IV into a supercomplex in intact yeast mitochondriaMitochondrial outer membrane permeabilization during apoptosis: the innocent bystander scenario.Cytochrome c adsorption to supported, anionic lipid bilayers studied via atomic force microscopy.Mitochondria in apoptosis: Bcl-2 family members and mitochondrial dynamics.Reactive oxygen species in mitochondria-mediated cell death.Cardiolipin or MTCH2 can serve as tBID receptors during apoptosisMitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formationLinkage between mitochondrial hypovirulence and viral hypovirulence in the chestnut blight fungus revealed by cDNA microarray analysis.Death upon a kiss: mitochondrial outer membrane composition and organelle communication govern sensitivity to BAK/BAX-dependent apoptosis.Lipids and cell death in yeast.Cloning and characterization of a cDNA encoding human cardiolipin synthase (hCLS1).Physiological and Pharmacological Control of BAK, BAX, and Beyond.Oxidative modification of cytochrome c by singlet oxygen.Yeast as a tool for studying proteins of the Bcl-2 familyCardiolipin and phosphatidylglycerol are not required for the in vivo action of Bcl-2 family proteins.Caspase-2 permeabilizes the outer mitochondrial membrane and disrupts the binding of cytochrome c to anionic phospholipids.Cytochrome c release from rat liver mitochondria is compromised by increased saturated cardiolipin species induced by sucrose feeding.Contributions to Bax insertion and oligomerization of lipids of the mitochondrial outer membrane.Lipidic pore formation by the concerted action of proapoptotic BAX and tBID.Mitochondrial Release of Pro-apoptotic Proteins
P2860
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P2860
Cardiolipin is not required for Bax-mediated cytochrome c release from yeast mitochondria.
description
2003 nî lūn-bûn
@nan
2003年の論文
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2003年論文
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2003年論文
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2003年論文
@zh-hk
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name
Cardiolipin is not required fo ...... lease from yeast mitochondria.
@en
type
label
Cardiolipin is not required fo ...... lease from yeast mitochondria.
@en
prefLabel
Cardiolipin is not required fo ...... lease from yeast mitochondria.
@en
P2093
P2860
P356
P1476
Cardiolipin is not required fo ...... lease from yeast mitochondria.
@en
P2093
Mari Enoksson
Martin Ott
Suzanne L Iverson
Vladimir Gogvadze
P2860
P304
P356
10.1074/JBC.M305020200
P407
P577
2003-10-08T00:00:00Z