Gene expression profiling of minimally differentiated acute myeloid leukemia: M0 is a distinct entity subdivided by RUNX1 mutation status.
about
RUNX1 mutations are associated with poor outcome in younger and older patients with cytogenetically normal acute myeloid leukemia and with distinct gene and MicroRNA expression signatures.Reinventing diagnostics for personalized therapy in oncologyRUNX1 truncation resulting from a cryptic and novel t(6;21)(q25;q22) chromosome translocation in acute myeloid leukemia: A case reportA mouse embryonic stem cell bank for inducible overexpression of human chromosome 21 genes.Runx1 and Cbfβ regulate the development of Flt3+ dendritic cell progenitors and restrict myeloproliferative disorder.TdT expression in acute myeloid leukemia with minimal differentiation is associated with distinctive clinicopathological features and better overall survival following stem cell transplantation.CCAAT/Enhancer Binding Protein-delta (C/EBP-delta) regulates cell growth, migration and differentiationRUNX1 repression-independent mechanisms of leukemogenesis by fusion genes CBFB-MYH11 and AML1-ETO (RUNX1-RUNX1T1).The prognostic significance of IRF8 transcripts in adult patients with acute myeloid leukemiaRUNX1 mutations in clonal myeloid disorders: from conventional cytogenetics to next generation sequencing, a story 40 years in the makingGene and protein analysis reveals that p53 pathway is functionally inactivated in cytogenetically normal Acute Myeloid Leukemia and Acute Promyelocytic LeukemiaRUNX1 mutations in cytogenetically normal acute myeloid leukemia are associated with a poor prognosis and up-regulation of lymphoid genes.Genome wide molecular analysis of minimally differentiated acute myeloid leukemia.Genome-scale technology driven advances to research into normal and malignant haematopoiesis.Expression of CEBPA is reduced in RUNX1-mutated acute myeloid leukemia.RUNX1 and CBFβ Mutations and Activities of Their Wild-Type Alleles in AML.Molecular mechanisms that produce secondary MDS/AML by RUNX1/AML1 point mutations.
P2860
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P2860
Gene expression profiling of minimally differentiated acute myeloid leukemia: M0 is a distinct entity subdivided by RUNX1 mutation status.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Gene expression profiling of m ...... ided by RUNX1 mutation status.
@en
type
label
Gene expression profiling of m ...... ided by RUNX1 mutation status.
@en
prefLabel
Gene expression profiling of m ...... ided by RUNX1 mutation status.
@en
P2093
P1433
P1476
Gene expression profiling of m ...... vided by RUNX1 mutation status
@en
P2093
Bas J Wouters
Claudia Erpelinck-Verschueren
Micheline Giphart-Gassler
Peter J M Valk
Peter van der Spek
Ruud Delwel
Sigrid M A Swagemakers
P304
P356
10.1182/BLOOD-2009-03-211334
P407
P577
2009-08-07T00:00:00Z