Resistance to 2,3,7,8-tetrachlorodibenzo-p-dioxin toxicity and abnormal liver development in mice carrying a mutation in the nuclear localization sequence of the aryl hydrocarbon receptor.
about
The aryl hydrocarbon receptor: a perspective on potential roles in the immune systemSubchronic exposure to TCDD, PeCDF, PCB126, and PCB153: effect on hepatic gene expressionAryl hydrocarbon receptor-dependent liver development and hepatotoxicity are mediated by different cell types.Repression of aryl hydrocarbon receptor (AHR) signaling by AHR repressor: role of DNA binding and competition for AHR nuclear translocatorThe aryl hydrocarbon receptor (AHR) transcription factor regulates megakaryocytic polyploidizationEpigenetics and environmental chemicalsRelB, a new partner of aryl hydrocarbon receptor-mediated transcriptionGene-chemical interactions in the developing mammalian nervous system: Effects on proliferation, neurogenesis and differentiationThe aryl hydrocarbon receptor complex and the control of gene expressionExactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptorSU5416, a VEGF receptor inhibitor and ligand of the AHR, represents a new alternative for immunomodulationTranscriptomic responses to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in liver: comparison of rat and mouse.Aryl hydrocarbon receptor (AHR)-regulated transcriptomic changes in rats sensitive or resistant to major dioxin toxicities.Arylhydrocarbon receptor activation in NCI-H441 cells and C57BL/6 mice: possible mechanisms for lung dysfunctionNAD+ loss, a new player in AhR biology: prevention of thymus atrophy and hepatosteatosis by NAD+ repletion.Structural hierarchy controlling dimerization and target DNA recognition in the AHR transcriptional complexAn activated renin-angiotensin system maintains normal blood pressure in aryl hydrocarbon receptor heterozygous mice but not in null mice.Aryl hydrocarbon receptor activation by cAMP vs. dioxin: divergent signaling pathways.The aryl hydrocarbon receptor-interacting protein (AIP) is required for dioxin-induced hepatotoxicity but not for the induction of the Cyp1a1 and Cyp1a2 genesAryl hydrocarbon receptor nuclear translocator in hepatocytes is required for aryl hydrocarbon receptor-mediated adaptive and toxic responses in liverXenobiotic metabolism, disposition, and regulation by receptors: from biochemical phenomenon to predictors of major toxicitiesCross-species transcriptomic analysis elucidates constitutive aryl hydrocarbon receptor activityAhR deficiency impairs expression of LPS-induced inflammatory genes in miceAryl hydrocarbon receptor-mediated induction of Stearoyl-CoA desaturase 1 alters hepatic fatty acid composition in TCDD-elicited steatosisAdvances in analytical techniques for polychlorinated dibenzo-p-dioxins, polychlorinated dibenzofurans and dioxin-like PCBsLoss of the Mono-ADP-ribosyltransferase, Tiparp, Increases Sensitivity to Dioxin-induced Steatohepatitis and LethalityPeroxisome proliferators and receptor-mediated hepatic carcinogenesis.Deletion or activation of the aryl hydrocarbon receptor alters adult hippocampal neurogenesis and contextual fear memory.Male and female mice show significant differences in hepatic transcriptomic response to 2,3,7,8-tetrachlorodibenzo-p-dioxin.Compendium of TCDD-mediated transcriptomic response datasets in mammalian model systems.Novel cellular targets of AhR underlie alterations in neutrophilic inflammation and inducible nitric oxide synthase expression during influenza virus infection.Aryl Hydrocarbon Receptor Activation Synergistically Induces Lipopolysaccharide-Mediated Expression of Proinflammatory Chemokine (c-c motif) Ligand 20.Complexities in understanding the nature of the dose-response for dioxins and related compounds.Hepatic transcriptional networks induced by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin.Genomic profiling in nuclear receptor-mediated toxicity.Abnormal liver development and resistance to 2,3,7,8-tetrachlorodibenzo-p-dioxin toxicity in mice carrying a mutation in the DNA-binding domain of the aryl hydrocarbon receptor.Ah receptor binding to its cognate response element is required for dioxin-mediated toxicity.Environmental chemicals and microRNAs.The role of the dioxin-responsive element cluster between the Cyp1a1 and Cyp1a2 loci in aryl hydrocarbon receptor biology.Conserved genomic structure of the Cyp1a1 and Cyp1a2 loci and their dioxin responsive elements cluster
P2860
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P2860
Resistance to 2,3,7,8-tetrachlorodibenzo-p-dioxin toxicity and abnormal liver development in mice carrying a mutation in the nuclear localization sequence of the aryl hydrocarbon receptor.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Resistance to 2,3,7,8-tetrachl ...... the aryl hydrocarbon receptor.
@en
type
label
Resistance to 2,3,7,8-tetrachl ...... the aryl hydrocarbon receptor.
@en
prefLabel
Resistance to 2,3,7,8-tetrachl ...... the aryl hydrocarbon receptor.
@en
P2093
P2860
P356
P1476
Resistance to 2,3,7,8-tetrachl ...... the aryl hydrocarbon receptor
@en
P2093
Bernice C Lin
Christopher A Bradfield
Edward Glover
Maureen K Bunger
Susan M Moran
Tami L Thomae
P2860
P304
17767-17774
P356
10.1074/JBC.M209594200
P407
P577
2003-03-05T00:00:00Z