Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia. - Wikidata - wikimedia - TriplyDB

Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

http://www.wikidata.org/entity/Q38357013

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Alterations in postnatal neurogenesis and dopamine dysregulation in schizophrenia: a hypothesis Identification of neuronal nuclei (NeuN) as Fox-3, a new member of the Fox-1 gene family of splicing factors High-Field MRI Reveals a Drastic Increase of Hypoxia-Induced Microhemorrhages upon Tissue Reoxygenation in the Mouse Brain with Strong Predominance in the Olfactory Bulb Directing astroglia from the cerebral cortex into subtype specific functional neurons Quantitative assessment of fibroblast growth factor receptor 1 expression in neurons and glia.Recent advances in Tourette syndrome.Neurobiology of premature brain injury.Combustion smoke-induced inflammation in the olfactory bulb of adult rats.Genome Resequencing Identifies Unique Adaptations of Tibetan Chickens to Hypoxia and High-Dose Ultraviolet Radiation in High-Altitude Environments.Toward a novel endogenous anxiolytic factor, fibroblast growth factor 2.Microstructural and functional connectivity in the developing preterm brain.Cortical glial fibrillary acidic protein-positive cells generate neurons after perinatal hypoxic injury.How animal models inform child and adolescent psychiatry.Abbreviated exposure to hypoxia is sufficient to induce CNS dysmyelination, modulate spinal motor neuron composition, and impair motor development in neonatal mice Maternal immune activation by LPS selectively alters specific gene expression profiles of interneuron migration and oxidative stress in the fetus without triggering a fetal immune response Selective reduction of cerebral cortex GABA neurons in a late gestation model of fetal alcohol spectrum disorder.Contribution of maternal oxygenic state to the effects of chronic postnatal hypoxia on mouse body and brain development.Oligodendrocyte regeneration after neonatal hypoxia requires FoxO1-mediated p27Kip1 expression.Newborn cortical neurons: only for neonates?Neurogenesis continues in the third trimester of pregnancy and is suppressed by premature birth.Hypoxia-induced developmental delays of inhibitory interneurons are reversed by environmental enrichment in the postnatal mouse forebrain Convergence of genetic and environmental factors on parvalbumin-positive interneurons in schizophrenia.Sex differences in a hypoxia model of preterm brain damage.Modeling premature brain injury and recovery.Sirt1 regulates glial progenitor proliferation and regeneration in white matter after neonatal brain injury.Inflammatory regulators of redirected neural migration in the injured brain.Fibroblast growth factors in neurodevelopment and psychopathology.The endogenous regenerative capacity of the damaged newborn brain: boosting neurogenesis with mesenchymal stem cell treatment.Neurogenesis and maturation in neonatal brain injury.Controversies in preterm brain injury.Postnatal Neural Stem Cells.Exciting news from the adult mouse subventricular zone.Dysfunction in Motor Coordination in Neonatal White Matter Injury Model Without Apparent Neuron Loss.Subventricular zone-derived oligodendrogenesis in injured neonatal white matter in mice enhanced by a nonerythropoietic erythropoietin derivative.Adult neurogenesis in the human striatum: possible implications for psychiatric disorders.Maternal high-fat diet influences outcomes after neonatal hypoxic-ischemic brain injury in rodents.Analysis of 182 cerebral palsy transcriptomes points to dysregulation of trophic signalling pathways and overlap with autism.Hypoxia Delays Oligodendrocyte Progenitor Cell Migration and Myelin Formation by Suppressing Bmp2b Signaling in Larval Zebrafish

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Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

http://www.wikidata.org/entity/Q38357013

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2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

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type

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Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

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prefLabel

Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

@en

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JNEUROSCI.4516-08.2009

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Journal of Neuroscience

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Fgfr1 is required for cortical regeneration and repair after perinatal hypoxia.

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P2093

Devon M Fagel

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Elise Cheng

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John Silbereis

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Laura R Ment

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Yasushi Ohkubo

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Yosif Ganat

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P2860

Neuronal defects and delayed wound healing in mice lacking fibroblast growth factor 2

T-brain-1: a homolog of Brachyury whose expression defines molecularly distinct domains within the cerebral cortex

Cell proliferation without neurogenesis in adult primate neocortex

Regeneration of hippocampal pyramidal neurons after ischemic brain injury by recruitment of endogenous neural progenitors

Notch signalling regulates stem cell numbers in vitro and in vivo

Restricted proliferation and migration of postnatally generated neurons derived from the forebrain subventricular zone

Origin and progeny of reactive gliosis: A source of multipotent cells in the injured brain

Loss of glutamatergic pyramidal neurons in frontal and temporal cortex resulting from attenuation of FGFR1 signaling is associated with spontaneous hyperactivity in mice

FGFR1 is independently required in both developing mid- and hindbrain for sustained response to isthmic signals

Neuronal replacement from endogenous precursors in the adult brain after stroke

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Flora M. Vaccarino

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