Rapid, sequential activation of mitogen-activated protein kinases and transcription factors precedes proinflammatory cytokine mRNA expression in spleens of mice exposed to the trichothecene vomitoxin.
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Advances in deoxynivalenol toxicity mechanisms: the brain as a targetDeoxynivalenol as a new factor in the persistence of intestinal inflammatory diseases: an emerging hypothesis through possible modulation of Th17-mediated responseEnvironmental immune disruptors, inflammation and cancer riskAssessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge aheadCo-exposure to low doses of the food contaminants deoxynivalenol and nivalenol has a synergistic inflammatory effect on intestinal explants.Activation of the Classical Mitogen-Activated Protein Kinases Is Part of the Shiga Toxin-Induced Ribotoxic Stress Response and May Contribute to Shiga Toxin-Induced Inflammation.Therapeutic effects of glutamic acid in piglets challenged with deoxynivalenolEmbryotoxicity Caused by DON-Induced Oxidative Stress Mediated by Nrf2/HO-1 Pathway.Proteomic analysis of the effects of the immunomodulatory mycotoxin deoxynivalenol.Induction of apoptotic lesions in liver and lymphoid tissues and modulation of cytokine mRNA expression by acute exposure to deoxynivalenol in piglets.Bacillus subtilis Protects Porcine Intestinal Barrier from Deoxynivalenol via Improved Zonula Occludens-1 Expression.n-3 polyunsaturated fatty acids and autoimmune-mediated glomerulonephritis.Suppression of insulin-like growth factor acid-labile subunit expression--a novel mechanism for deoxynivalenol-induced growth retardationStachybotrys chartarum, trichothecene mycotoxins, and damp building-related illness: new insights into a public health enigma.Mechanisms of deoxynivalenol-induced gene expression and apoptosis.Effects of trichothecene mycotoxins on eukaryotic cells: a review.Satratoxin G interaction with 40S and 60S ribosomal subunits precedes apoptosis in the macrophage.Deoxynivalenol-induced proinflammatory gene expression: mechanisms and pathological sequelae.Effects of oral exposure to naturally-occurring and synthetic deoxynivalenol congeners on proinflammatory cytokine and chemokine mRNA expression in the mouse.Zearalenone mycotoxin affects immune mediators, MAPK signalling molecules, nuclear receptors and genome-wide gene expression in pig spleen.The Food Contaminant Mycotoxin Deoxynivalenol Inhibits the Swallowing Reflex in Anaesthetized Rats.Nivalenol has a greater impact than deoxynivalenol on pig jejunum mucosa in vitro on explants and in vivo on intestinal loopsTargets and intracellular signaling mechanisms for deoxynivalenol-induced ribosomal RNA cleavage.Deoxynivalenol: toxicology and potential effects on humans.Shiga toxin 2-induced intestinal pathology in infant rabbits is A-subunit dependent and responsive to the tyrosine kinase and potential ZAK inhibitor imatinibModulation of inflammatory gene expression by the ribotoxin deoxynivalenol involves coordinate regulation of the transcriptome and translatomeTissue distribution and proinflammatory cytokine gene expression following acute oral exposure to deoxynivalenol: comparison of weanling and adult mice.Early phosphoproteomic changes in the mouse spleen during deoxynivalenol-induced ribotoxic stressImmunochemical assessment of deoxynivalenol tissue distribution following oral exposure in the mouse.A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.Comparative induction of 28S ribosomal RNA cleavage by ricin and the trichothecenes deoxynivalenol and T-2 toxin in the macrophage.Induction of suppressors of cytokine signaling by the trichothecene deoxynivalenol in the mouse.Dynamic changes in ribosome-associated proteome and phosphoproteome during deoxynivalenol-induced translation inhibition and ribotoxic stressImpacts of the feed contaminant deoxynivalenol on the intestine of monogastric animals: poultry and swine.Gene expression profiling in spleens of deoxynivalenol-exposed mice: immediate early genes as primary targets.The Putative Role of Viruses, Bacteria, and Chronic Fungal Biotoxin Exposure in the Genesis of Intractable Fatigue Accompanied by Cognitive and Physical Disability.Deoxynivalenol and its metabolite deepoxy-deoxynivalenol: multi-parameter analysis for the evaluation of cytotoxicity and cellular effects.Endosulfan induces COX-2 expression via NADPH oxidase and the ROS, MAPK, and Akt pathways.Characterization of the modes of action of deoxynivalenol (DON) in the human Jurkat T-cell line.Influence of temperature and pH on the degradation of deoxynivalenol (DON) in aqueous medium: comparative cytotoxicity of DON and degraded product.
P2860
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P2860
Rapid, sequential activation of mitogen-activated protein kinases and transcription factors precedes proinflammatory cytokine mRNA expression in spleens of mice exposed to the trichothecene vomitoxin.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Rapid, sequential activation o ...... o the trichothecene vomitoxin.
@en
type
label
Rapid, sequential activation o ...... o the trichothecene vomitoxin.
@en
prefLabel
Rapid, sequential activation o ...... o the trichothecene vomitoxin.
@en
P2093
P356
P1476
Rapid, sequential activation o ...... o the trichothecene vomitoxin.
@en
P2093
Hui-Ren Zhou
James J Pestka
Zahidul Islam
P304
P356
10.1093/TOXSCI/KFG006
P577
2003-03-01T00:00:00Z