Epstein-Barr virus infection induces aberrant TLR activation pathway and fibroblast-myofibroblast conversion in scleroderma
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Current perspectives on the immunopathogenesis of systemic sclerosisHCV derived from sera of HCV-infected patients induces pro-fibrotic effects in human primary fibroblasts by activating GLI2Toll-like receptors in the pathogenesis of autoimmune diseases: recent and emerging translational developmentsThe role of toll like receptors in giant cell arteritisInfections as a cause of autoimmune rheumatic diseasesGene expression profiling offers insights into the role of innate immune signaling in SScAnimal models of systemic sclerosis: their utility and limitations.Systems level analysis of systemic sclerosis shows a network of immune and profibrotic pathways connected with genetic polymorphisms.Infectomics and autoinfectomics: a tool to study infectious-induced autoimmunity.Tenascin-C drives persistence of organ fibrosis.Epstein-Barr virus lytic infection promotes activation of Toll-like receptor 8 innate immune response in systemic sclerosis monocytes.A novel multi-network approach reveals tissue-specific cellular modulators of fibrosis in systemic sclerosis.Emerging roles of innate immune signaling and toll-like receptors in fibrosis and systemic sclerosis.Toll-Like Receptor Pathways in Autoimmune Diseases.Role of innate immune system in systemic sclerosis.Serological evidence that activation of ubiquitous human herpesvirus-6 (HHV-6) plays a role in chronic idiopathic/spontaneous urticaria (CIU).Fresh Insights into Disease Etiology and the Role of Microbial Pathogens.An oligodeoxynucleotide with AAAG repeats significantly attenuates burn-induced systemic inflammatory responses via inhibiting interferon regulatory factor 5 pathwayIdiopathic nephrotic syndrome: the EBV hypothesis.The significance of macrophage polarization subtypes for animal models of tissue fibrosis and human fibrotic diseasesMicrobial infection, inflammation and epithelial ovarian cancer.Gene sharing between Epstein-Barr virus and human immune response genes.Toll-like receptor 9 in systemic sclerosis patients: relation to modified Rodnan skin score, disease severity, and functional status.Unmet Needs in Systemic Sclerosis Understanding and Treatment: the Knowledge Gaps from a Scientist's, Clinician's, and Patient's Perspective.Toll-like Receptor 9 Signaling Is Augmented in Systemic Sclerosis and Elicits Transforming Growth Factor β-Dependent Fibroblast Activation.Immune complexes containing scleroderma-specific autoantibodies induce a profibrotic and proinflammatory phenotype in skin fibroblasts
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P2860
Epstein-Barr virus infection induces aberrant TLR activation pathway and fibroblast-myofibroblast conversion in scleroderma
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Epstein-Barr virus infection i ...... last conversion in scleroderma
@en
type
label
Epstein-Barr virus infection i ...... last conversion in scleroderma
@en
prefLabel
Epstein-Barr virus infection i ...... last conversion in scleroderma
@en
P2093
P2860
P356
P1476
Epstein-Barr virus infection i ...... last conversion in scleroderma
@en
P2093
Alberto Faggioni
Cristina Padilla
Giuseppina A Farina
Mara Cirone
Maria Trojanowska
Michael York
Robert Lafyatis
Sarah McLaughlin
P2860
P2888
P304
P356
10.1038/JID.2013.423
P407
P577
2013-10-11T00:00:00Z
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P6179
1049578484