Expression and function of C/EBP homology protein (GADD153) in podocytes. - Wikidata - wikimedia - TriplyDB

Expression and function of C/EBP homology protein (GADD153) in podocytes.

Expression and function of C/EBP homology protein (GADD153) in podocytes.

http://www.wikidata.org/entity/Q38404766

about

Endoplasmic reticulum stress and unfolded protein response in renal pathophysiology: Janus faces Regulation of podocyte survival and endoplasmic reticulum stress by fatty acids PKR-dependent CHOP induction limits hyperoxia-induced lung injury.The endoplasmic reticulum stress response and diabetic kidney disease.Angiotensin II induces endoplasmic reticulum stress in podocyte, which would be further augmented by PI3-kinase inhibition C/EBP homologous protein (CHOP) contributes to suppression of metabolic genes during endoplasmic reticulum stress in the liver.Endoplasmic reticulum stress plays a key role in the pathogenesis of diabetic peripheral neuropathy Positive role of CCAAT/enhancer-binding protein homologous protein, a transcription factor involved in the endoplasmic reticulum stress response in the development of colitis C/EBP homologous protein (CHOP) deficiency ameliorates renal fibrosis in unilateral ureteral obstructive kidney disease.C/EBP homologous protein deficiency aggravates acute pancreatitis and associated lung injury Diabetes-Induced Reactive Oxygen Species: Mechanism of Their Generation and Role in Renal Injury Mitochondrial reactive oxygen species as a mystery voice in hepatitis C.Evidence for activation of the unfolded protein response in collagen IV nephropathies Mesencephalic astrocyte-derived neurotrophic factor (MANF), a new player in endoplasmic reticulum diseases: structure, biology, and therapeutic roles.Mesencephalic Astrocyte-Derived Neurotrophic Factor as a Urine Biomarker for Endoplasmic Reticulum Stress-Related Kidney Diseases.Endoplasmic reticulum stress inhibition attenuates hypertensive chronic kidney disease through reduction in proteinuria.TRB3 is stimulated in diabetic kidneys, regulated by the ER stress marker CHOP, and is a suppressor of podocyte MCP-1 Albumin overload down-regulates integrin-β1 through reactive oxygen species-endoplasmic reticulum stress pathway in podocytes.GDC-0879, a BRAFV600E Inhibitor, Protects Kidney Podocytes from Death.Elevated urinary CRELD2 is associated with endoplasmic reticulum stress-mediated kidney disease.Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases.Bestrophin-3 is differently expressed in normal and injured mouse glomerular podocytes.Endoplasmic reticulum stress inhibition limits the progression of chronic kidney disease in the Dahl salt-sensitive rat.Ursodeoxycholic acid and 4-phenylbutyrate prevent endoplasmic reticulum stress-induced podocyte apoptosis in diabetic nephropathy.Enhanced apoptotic and reduced protective response in chondrocytes following endoplasmic reticulum stress in osteoarthritic cartilage.

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P2860

Expression and function of C/EBP homology protein (GADD153) in podocytes.

http://www.wikidata.org/entity/Q38404766

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

Expression and function of C/EBP homology protein (GADD153) in podocytes.

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type

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Expression and function of C/EBP homology protein (GADD153) in podocytes.

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Expression and function of C/EBP homology protein (GADD153) in podocytes.

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AJPATH.2006.040774

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The American Journal of Pathology

P1476

Expression and function of C/EBP homology protein (GADD153) in podocytes.

@en

P2093

Albrecht Schwab

http://www.w3.org/2001/XMLSchema#string

Barbara Müller

http://www.w3.org/2001/XMLSchema#string

Christian August

http://www.w3.org/2001/XMLSchema#string

Detlef Lang

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Erik Springer

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Hermann Pavenstädt

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Martin F Bek

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Michael Bayer

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Rolf Rohrbach

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Stefan Greiber

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P2860

Classification of gas5 as a multi-small-nucleolar-RNA (snoRNA) host gene and a member of the 5'-terminal oligopyrimidine gene family reveals common features of snoRNA host genes.

Identification of novel stress-induced genes downstream of chop

Stress-induced phosphorylation and activation of the transcription factor CHOP (GADD153) by p38 MAP Kinase

CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription

CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bcl2 and perturbing the cellular redox state

Expression of the pro-apoptotic gene gadd153/chop is elevated in liver with aging and sensitizes cells to oxidant injury

CHOP-Dependent stress-inducible expression of a novel form of carbonic anhydrase VI

Protection of alpha(3) integrin-mediated podocyte shape by superoxide dismutase in the puromycin aminonucleoside nephrosis rat.

Roles of the podocyte in glomerular function.

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