Third trimester-equivalent ethanol exposure is characterized by an acute cellular stress response and an ontogenetic disruption of genes critical for synaptic establishment and function in mice.
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Epigenetic Mechanisms in Developmental Alcohol-Induced Neurobehavioral DeficitsRecurrent Moderate Hypoglycemia Suppresses Brain-Derived Neurotrophic Factor Expression in the Prefrontal Cortex and Impairs Sensorimotor Gating in the Posthypoglycemic Period in Young Rats.Chromatin Switches during Neural Cell Differentiation and Their Dysregulation by Prenatal Alcohol ExposureDevelopmental alcohol exposure leads to a persistent change on astrocyte secretome.Influence of sex and stress exposure across the lifespan on endophenotypes of depression: focus on behavior, glucocorticoids, and hippocampus.Alteration of Gene Expression, DNA Methylation, and Histone Methylation in Free Radical Scavenging Networks in Adult Mouse Hippocampus following Fetal Alcohol Exposure.Revisiting Chaos Theorem to Understand the Nature of miRNAs in Response to Drugs of Abuse.Epigenetics studies of fetal alcohol spectrum disorder: where are we now?Associative DNA methylation changes in children with prenatal alcohol exposure.Heterogeneity of p53 dependent genomic responses following ethanol exposure in a developmental mouse model of fetal alcohol spectrum disorder.
P2860
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P2860
Third trimester-equivalent ethanol exposure is characterized by an acute cellular stress response and an ontogenetic disruption of genes critical for synaptic establishment and function in mice.
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2014 nî lūn-bûn
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2014年の論文
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name
Third trimester-equivalent eth ...... lishment and function in mice.
@en
type
label
Third trimester-equivalent eth ...... lishment and function in mice.
@en
prefLabel
Third trimester-equivalent eth ...... lishment and function in mice.
@en
P356
P1476
Third trimester-equivalent eth ...... lishment and function in mice.
@en
P2093
Randa L Stringer
Shiva M Singh
P304
P356
10.1159/000365549
P577
2014-09-30T00:00:00Z