Chromosomal imbalances in primary and metastatic melanomas revealed by comparative genomic hybridization.
about
ASEQ: fast allele-specific studies from next-generation sequencing data.Fractal dimension of chromatin is an independent prognostic factor for survival in melanoma.Superficial spreading and nodular melanoma are distinct biological entities: a challenge to the linear progression model.Chromosomal aberrations related to metastasis of human solid tumorsIn-silico identification and functional validation of allele-dependent AR enhancers.Chromosome 6p amplification and cancer progression.Immunohistochemistry in melanocytic proliferative lesions.Stem cells in melanoma developmentDistinguishing melanocytic nevi from melanoma by DNA copy number changes: comparative genomic hybridization as a research and diagnostic tool.Putative cancer stem cells in cutaneous malignancies.Cancer stem cells and human malignant melanomaFluorescence in-situ hybridization analysis for melanoma diagnosis.The 11q13-q14 amplicon: clinicopathological correlations and potential drivers.The ectodomain shedding of E-cadherin by ADAM15 supports ErbB receptor activation.Copy number variation in archival melanoma biopsies versus benign melanocytic lesions.A nonredundant multicolor bar code as a screening tool for rearrangements in neoplasia.Genomic and transcriptomic profiling of resistant CEM/ADR-5000 and sensitive CCRF-CEM leukaemia cells for unravelling the full complexity of multi-factorial multidrug resistanceCAS (CSE1L) signaling pathway in tumor progression and its potential as a biomarker and target for targeted therapy.Distinct MHC gene expression patterns during progression of melanoma.Involvement of overexpressed wild-type BRAF in the growth of malignant melanoma cell lines.Concurrence of chromosome 3 and 4 aberrations in human uveal melanoma.IL-10 expression by primary tumor cells correlates with melanoma progression from radial to vertical growth phase and development of metastatic competenceUV-B-type mutations and chromosomal imbalances indicate common pathways for the development of Merkel and skin squamous cell carcinomas.Extra copies of c-myc are more pronounced in nodular melanomas than in superficial spreading melanomas as revealed by fluorescence in situ hybridisation.Characterization of candidate gene copy number alterations in the 11q13 region along with BRAF and NRAS mutations in human melanoma.Whole genome sequencing puts forward hypotheses on metastasis evolution and therapy in colorectal cancer
P2860
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P2860
Chromosomal imbalances in primary and metastatic melanomas revealed by comparative genomic hybridization.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Chromosomal imbalances in prim ...... arative genomic hybridization.
@en
type
label
Chromosomal imbalances in prim ...... arative genomic hybridization.
@en
prefLabel
Chromosomal imbalances in prim ...... arative genomic hybridization.
@en
P2093
P2860
P356
P1433
P1476
Chromosomal imbalances in prim ...... arative genomic hybridization.
@en
P2093
P2860
P304
P356
10.1002/CYTO.1131
P577
2001-08-01T00:00:00Z