Mesenchymal stromal cells to halt the progression of type 1 diabetes? - Wikidata - wikimedia - TriplyDB

Mesenchymal stromal cells to halt the progression of type 1 diabetes?

Mesenchymal stromal cells to halt the progression of type 1 diabetes?

http://www.wikidata.org/entity/Q38499617

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Infusion with Human Bone Marrow-derived Mesenchymal Stem Cells Improves β-cell Function in Patients and Non-obese Mice with Severe Diabetes An update clinical application of amniotic fluid-derived stem cells (AFSCs) in cancer cell therapy and tissue engineering.Mesenchymal Stem Cells as New Therapeutic Approach for Diabetes and Pancreatic Disorders

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Mesenchymal stromal cells to halt the progression of type 1 diabetes?

http://www.wikidata.org/entity/Q38499617

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

@zh-tw

2015年论文

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2015年论文

@zh

2015年论文

@zh-cn

name

Mesenchymal stromal cells to halt the progression of type 1 diabetes?

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Mesenchymal stromal cells to halt the progression of type 1 diabetes?

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Mesenchymal stromal cells to halt the progression of type 1 diabetes?

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S11892-015-0616-3

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Current diabetes report

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Mesenchymal stromal cells to halt the progression of type 1 diabetes?

@en

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Katarina Le Blanc

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Per-Ola Carlsson

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Proinflammatory stimuli induce galectin-9 in human mesenchymal stromal cells to suppress T-cell proliferation

Type 1 diabetes

Minimal criteria for defining multipotent mesenchymal stromal cells. The International Society for Cellular Therapy position statement

Therapeutic benefit of intravenous administration of bone marrow stromal cells after cerebral ischemia in rats

Human mesenchymal stem cells modulate allogeneic immune cell responses

Autologous nonmyeloablative hematopoietic stem cell transplantation in newly diagnosed type 1 diabetes mellitus

Mesenchymal stem cells for treatment of steroid-resistant, severe, acute graft-versus-host disease: a phase II study

Treatment of severe acute graft-versus-host disease with third party haploidentical mesenchymal stem cells

Bone marrow stromal cells attenuate sepsis via prostaglandin E(2)-dependent reprogramming of host macrophages to increase their interleukin-10 production

Intravenous hMSCs improve myocardial infarction in mice because cells embolized in lung are activated to secrete the anti-inflammatory protein TSG-6

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s11892-015-0616-3

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scholarly article

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10.1007/S11892-015-0616-3

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26003192

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type-1 diabetes