Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.
about
Circadian Clocks in Articular Cartilage and Bone: A Compass in the Sea of Matrices.The Role of Circadian Rhythms in Muscular and Osseous Physiology and Their Regulation by Nutrition and Exercise.The brain-joint axis in osteoarthritis: nerves, circadian clocks and beyond.Signaling pathways regulating cartilage growth plate formation and activity.Integrative analysis of GWAS, eQTLs and meQTLs data suggests that multiple gene sets are associated with bone mineral density.BMAL1 Deficiency Contributes to Mandibular Dysplasia by Upregulating MMP3.The importance of the circadian system & sleep for bone health.The Circadian Gene Clock Regulates Bone Formation Via PDIA3.The Interaction between Bmal1 and Per2 in Mouse BMSC Osteogenic Differentiation.Adult stem cell maintenance and tissue regeneration around the clock: do impaired stem cell clocks drive age-associated tissue degeneration?Insights into the Role of Circadian Rhythms in Bone Metabolism: A Promising Intervention Target?
P2860
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P2860
Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.
@en
type
label
Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.
@en
prefLabel
Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.
@en
P2093
P2860
P1433
P1476
Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype.
@en
P2093
Amit Vasanji
Ronald J Midura
William E Samsa
P2860
P304
P356
10.1016/J.BONE.2016.01.006
P577
2016-01-11T00:00:00Z