Pathogenetic significance of increased levels of interleukin-8 in the peritoneal fluid of patients with endometriosis.
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Immunological aspects of endometriosis: a reviewLaser capture microdissection and cDNA array analysis of endometrium identify CCL16 and CCL21 as epithelial-derived inflammatory mediators associated with endometriosisEndometrial quality in infertile women with endometriosis.Inhibitor of apoptosis proteins (IAPs) may be effective therapeutic targets for treating endometriosis.Reciprocal communication between endometrial stromal cells and macrophages.The role of cytokines in endometriosis.Immunology and endometriosis.Obstetrical Complications in Women with Endometriosis: A Cohort Study in JapanThe role of endometrium in endometriosis.The remedial effect of soluble interleukin-1 receptor type II on endometriosis in the nude mouse model.In vitro and in vivo approaches to study angiogenesis in the pathophysiology and therapy of endometriosis.Angiogenesis and endometriosisParacrine regulation of endometriotic tissue.Thrombospondin-1 serum levels do not correlate with pelvic pain in patients with ovarian endometriosis.Pathophysiological roles of chemokines in human reproduction: an overview.Dose-response effect of interleukin (IL)-1β, tumour necrosis factor (TNF)-α, and interferon-γ on the in vitro production of epithelial neutrophil activating peptide-78 (ENA-78), IL-8, and IL-6 by human endometrial stromal cells.Toll-like receptor (TLR) expression and TLR-mediated cytokine/chemokine production by human uterine epithelial cells.Chemokine receptor expression in human endometrium.Menstrual cycle-specific inhibition of endometrial stromal cell proliferation by oncostatin M.Drug-induced apoptosis was markedly attenuated in endometriotic stromal cells.Possible pathophysiological roles of mitogen-activated protein kinases (MAPKs) in endometriosis.Endometriotic cells are resistant to interferon-gamma-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis.Gonadotropin-releasing hormone analogues reduce the proliferation of endometrial stromal cells but not endometriotic cells.Clinical management of endometriosis-associated infertility.Novel augmentation by bufalin of protein kinase C-induced cyclooxygenase-2 and IL-8 production in human breast cancer cells.Inhibition of IAP (inhibitor of apoptosis) proteins represses inflammatory status via nuclear factor-kappa B pathway in murine endometriosis lesions.Cytokine profiles in serum and peritoneal fluid from infertile women with and without endometriosis.Lipopolysaccharide promotes the development of murine endometriosis-like lesions via the nuclear factor-kappa B pathway.Inhibition of dual specificity phosphatase-2 by hypoxia promotes interleukin-8-mediated angiogenesis in endometriosis.The cellular inhibitor of apoptosis protein-2 is a possible target of novel treatment for endometriosis.Increased Interleukin-6 Messenger RNA Expression in Macrophage-Cocultured Endometrial Stromal Cells in Adenomyosis
P2860
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P2860
Pathogenetic significance of increased levels of interleukin-8 in the peritoneal fluid of patients with endometriosis.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Pathogenetic significance of i ...... f patients with endometriosis.
@en
type
label
Pathogenetic significance of i ...... f patients with endometriosis.
@en
prefLabel
Pathogenetic significance of i ...... f patients with endometriosis.
@en
P2093
P1476
Pathogenetic significance of i ...... f patients with endometriosis.
@en
P2093
P304
P356
10.1016/S0015-0282(98)00049-1
P407
P577
1998-05-01T00:00:00Z