KEAP1-NRF2 signalling and autophagy in protection against oxidative and reductive proteotoxicity. - Wikidata - wikimedia - TriplyDB

KEAP1-NRF2 signalling and autophagy in protection against oxidative and reductive proteotoxicity.

KEAP1-NRF2 signalling and autophagy in protection against oxidative and reductive proteotoxicity.

http://www.wikidata.org/entity/Q38553032

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Antireduction: an ancient strategy fit for future Sulfation of the FLAG epitope is affected by co-expression of G protein-coupled receptors in a mammalian cell model Redox biology and the interface between bioenergetics, autophagy and circadian control of metabolism Nano-liquid Chromatography-orbitrap MS-based Quantitative Proteomics Reveals Differences Between the Mechanisms of Action of Carnosic Acid and Carnosol in Colon Cancer Cells.Nuclear Factor Erythroid 2-Related Factor 2 Deficiency Results in Amplification of the Liver Fat-Lowering Effect of Estrogen.A biphasic effect of TNF-α in regulation of the Keap1/Nrf2 pathway in cardiomyocytes.Transient Proteotoxicity of Bacterial Virulence Factor Pyocyanin in Renal Tubular Epithelial Cells Induces ER-Related Vacuolation and Can Be Efficiently Modulated by Iron Chelators.Differential regulation of autophagy and mitophagy in pulmonary diseases.Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin.Nrf2 targeting by sulforaphane: A potential therapy for cancer treatment.Transcription factor NFE2L2/NRF2 is a regulator of macroautophagy genes.The Role of the Nrf2/ARE Antioxidant System in Preventing Cardiovascular Diseases.Redox stress and signaling during vertebrate embryonic development: Regulation and responses.Measuring redox-active species in cells and tissues. Focus on "A case of mistaken identity: are reactive oxygen species actually reactive sulfide species?".KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease.Mitochondrial function and autophagy: integrating proteotoxic, redox, and metabolic stress in Parkinson's disease.Purification, Preliminary Characterization and Hepatoprotective Effects of Polysaccharides from Dandelion Root.Abrupt salinity stress induces oxidative stress via the Nrf2-Keap1 signaling pathway in large yellow croaker Pseudosciaena crocea.Redox Paradox: A novel approach to therapeutics-resistant cancer.Signaling Pathways Regulating Redox Balance in Cancer Metabolism.TRIM16 controls assembly and degradation of protein aggregates by modulating the p62-NRF2 axis and autophagy Biological Activities, Health Benefits, and Therapeutic Properties of Avenanthramides: From Skin Protection to Prevention and Treatment of Cerebrovascular Diseases

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P2860

KEAP1-NRF2 signalling and autophagy in protection against oxidative and reductive proteotoxicity.

http://www.wikidata.org/entity/Q38553032

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

KEAP1-NRF2 signalling and auto ...... and reductive proteotoxicity.

@en

type

label

KEAP1-NRF2 signalling and auto ...... and reductive proteotoxicity.

@en

prefLabel

KEAP1-NRF2 signalling and auto ...... and reductive proteotoxicity.

@en

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Biochemical Journal

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KEAP1-NRF2 signalling and auto ...... and reductive proteotoxicity.

@en

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Jianhua Zhang

http://www.w3.org/2001/XMLSchema#string

Matthew Dodson

http://www.w3.org/2001/XMLSchema#string

Matthew Redmann

http://www.w3.org/2001/XMLSchema#string

Namakkal S Rajasekaran

http://www.w3.org/2001/XMLSchema#string

Victor Darley-Usmar

http://www.w3.org/2001/XMLSchema#string

P2860

Role of Nrf2 in oxidative stress and toxicity

NRF2 cysteine residues are critical for oxidant/electrophile-sensing, Kelch-like ECH-associated protein-1-dependent ubiquitination-proteasomal degradation, and transcription activation

Endogenous HMGB1 regulates autophagy

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

p62/SQSTM1 is a target gene for transcription factor NRF2 and creates a positive feedback loop by inducing antioxidant response element-driven gene transcription

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex.

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

Autophagy, mitochondria and oxidative stress: cross-talk and redox signalling

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347-355

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scholarly article

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10.1042/BJ20150568

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3

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469

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2015-08-01T00:00:00Z

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26205490

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P932

5514546

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