Supercompetitor status of Drosophila Myc cells requires p53 as a fitness sensor to reprogram metabolism and promote viability.
about
Non-autonomous overgrowth by oncogenic niche cells: Cellular cooperation and competition in tumorigenesisOf flies, mice, and men: evolutionarily conserved tissue damage responses and agingSpontaneous Cell Competition in Immortalized Mammalian Cell LinesApoptosis in Cellular Society: Communication between Apoptotic Cells and Their NeighborsThe interplay between obesity and cancer: a fly viewThe Stearoyl-CoA Desaturase-1 (Desat1) in Drosophila cooperated with Myc to Induce Autophagy and Growth, a Potential New Link to Tumor SurvivalDrosophila Myc: A master regulator of cellular performance.Cell competition: winning out by losing notchp53 Enables metabolic fitness and self-renewal of nephron progenitor cellsCardiac deficiency of single cytochrome oxidase assembly factor scox induces p53-dependent apoptosis in a Drosophila cardiomyopathy modelCell Competition Modifies Adult Stem Cell and Tissue Population Dynamics in a JAK-STAT-Dependent MannerAntioxidant Role for Lipid Droplets in a Stem Cell Niche of DrosophilaMYC, Cell Competition, and Cell Death in Cancer: The Inseparable Triad.Accumulation of differentiating intestinal stem cell progenies drives tumorigenesisFunction of Lipid Storage Droplet 1 (Lsd1) in Wing Development of Drosophila melanogaster.An ancient defense system eliminates unfit cells from developing tissues during cell competition.Oncogene-directed alterations in cancer cell metabolism.Socializing with MYC: cell competition in development and as a model for premalignant cancer.Cell competition in vertebrate organ size regulation.The p53 control of apoptosis and proliferation: lessons from Drosophila.p53 Isoforms: Key Regulators of the Cell Fate Decision.Cell competition with normal epithelial cells promotes apical extrusion of transformed cells through metabolic changes.Apoptotic mechanisms during competition of ribosomal protein mutant cells: roles of the initiator caspases Dronc and Dream/Strica.MDCK cells expressing constitutively active Yes-associated protein (YAP) undergo apical extrusion depending on neighboring cell status.Genetic and Genomic Response to Selection for Food Consumption in Drosophila melanogaster.Tumor suppressor gene OSCP1/NOR1 regulates apoptosis, proliferation, differentiation, and ROS generation during eye development of Drosophila melanogaster.Cell size control - a mechanism for maintaining fitness and function.Physiological and pathological relevance of cell competition in fly to mammals.Mosaic Analysis in Drosophila.Mechanisms of cell competition emerging from Drosophila studies.Breaking Down Neighbors to Fuel Tumorigenesis.Myc inhibits JNK-mediated cell death in vivo.Drosophila as a Model to Study the Link between Metabolism and Cancer.p53 is required for brain growth but is dispensable for resistance to nutrient restriction during Drosophila larval development.P53 and mTOR signalling determine fitness selection through cell competition during early mouse embryonic development.ARE-mediated decay controls gene expression and cellular metabolism upon oxygen variations.The p53/Adipose-Tissue/Cancer Nexus
P2860
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P2860
Supercompetitor status of Drosophila Myc cells requires p53 as a fitness sensor to reprogram metabolism and promote viability.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Supercompetitor status of Dros ...... abolism and promote viability.
@en
type
label
Supercompetitor status of Dros ...... abolism and promote viability.
@en
prefLabel
Supercompetitor status of Dros ...... abolism and promote viability.
@en
P2093
P2860
P1433
P1476
Supercompetitor status of Dros ...... abolism and promote viability.
@en
P2093
Catarina M Quinzii
Claire de la Cova
D Christine Wu
Laura A Johnston
Marcello Ziosi
Nanami Senoo-Matsuda
P2860
P304
P356
10.1016/J.CMET.2014.01.012
P577
2014-02-20T00:00:00Z