Synergy between individual TNF-dependent functions determines granuloma performance for controlling Mycobacterium tuberculosis infection.
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Tuneable resolution as a systems biology approach for multi-scale, multi-compartment computational modelsComputational and Empirical Studies Predict Mycobacterium tuberculosis-Specific T Cells as a Biomarker for Infection Outcome.Identification of key processes that control tumor necrosis factor availability in a tuberculosis granulomaPhosphodiesterase 4 inhibition reduces innate immunity and improves isoniazid clearance of Mycobacterium tuberculosis in the lungs of infected miceAgent-Based Modeling in Systems PharmacologyIn silico evaluation and exploration of antibiotic tuberculosis treatment regimensA multi-scale approach to designing therapeutics for tuberculosisA computational tool integrating host immunity with antibiotic dynamics to study tuberculosis treatmentSystems Pharmacology Approach Toward the Design of Inhaled Formulations of Rifampicin and Isoniazid for Treatment of TuberculosisMacrophage polarization drives granuloma outcome during Mycobacterium tuberculosis infectionRelational grounding facilitates development of scientifically useful multiscale modelsThe spectrum of latent tuberculosis: rethinking the biology and intervention strategiesMultiscale computational modeling reveals a critical role for TNF-α receptor 1 dynamics in tuberculosis granuloma formation.Intracellular bacillary burden reflects a burst size for Mycobacterium tuberculosis in vivo.Signal regulatory protein alpha (SIRPalpha) cells in the adaptive response to ESAT-6/CFP-10 protein of tuberculous mycobacteria.Tuberculous optochiasmatic arachnoiditis: a devastating form of tuberculous meningitis.Tuberculosis: global approaches to a global disease.Latent tuberculosis infection: myths, models, and molecular mechanismsPreferential infection and depletion of Mycobacterium tuberculosis-specific CD4 T cells after HIV-1 infection.Polymorphisms in the interleukin 18 receptor 1 gene and tuberculosis susceptibility among Chinese.Natural transmission of Plasmodium berghei exacerbates chronic tuberculosis in an experimental co-infection modelBayesian approach to model CD137 signaling in human M. tuberculosis in vitro responses.Spartan: a comprehensive tool for understanding uncertainty in simulations of biological systemsHow tumour necrosis factor blockers interfere with tuberculosis immunity.The roles of immune memory and aging in protective immunity and endogenous reactivation of tuberculosis.Multi-scale modeling predicts a balance of tumor necrosis factor-α and interleukin-10 controls the granuloma environment during Mycobacterium tuberculosis infection.A Petri net model of granulomatous inflammation: implications for IL-10 mediated control of Leishmania donovani infection.Systems biology in immunology: a computational modeling perspective.A role for systems epidemiology in tuberculosis researchA Computational, Tissue-Realistic Model of Pressure Ulcer Formation in Individuals with Spinal Cord Injury.Differential risk of tuberculosis reactivation among anti-TNF therapies is due to drug binding kinetics and permeability.Epidemiological significance of the domestic black pig (Sus scrofa) in maintenance of bovine tuberculosis in SicilyAnalysis of Plasma Cytokine and Chemokine Profiles in Patients with and without Tuberculosis by Liquid Array-Based Multiplexed Immunoassays.NF-κB Signaling Dynamics Play a Key Role in Infection Control in Tuberculosis.Lung necrosis and neutrophils reflect common pathways of susceptibility to Mycobacterium tuberculosis in genetically diverse, immune-competent mice.Oxygen Modulates the Effectiveness of Granuloma Mediated Host Response to Mycobacterium tuberculosis: A Multiscale Computational Biology ApproachMultiscale Model of Mycobacterium tuberculosis Infection Maps Metabolite and Gene Perturbations to Granuloma Sterilization PredictionsComputational Modeling Predicts Simultaneous Targeting of Fibroblasts and Epithelial Cells Is Necessary for Treatment of Pulmonary Fibrosis.A hybrid multi-compartment model of granuloma formation and T cell priming in tuberculosis.Microenvironments in tuberculous granulomas are delineated by distinct populations of macrophage subsets and expression of nitric oxide synthase and arginase isoforms.
P2860
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P2860
Synergy between individual TNF-dependent functions determines granuloma performance for controlling Mycobacterium tuberculosis infection.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Synergy between individual TNF ...... terium tuberculosis infection.
@en
type
label
Synergy between individual TNF ...... terium tuberculosis infection.
@en
prefLabel
Synergy between individual TNF ...... terium tuberculosis infection.
@en
P2860
P356
P1476
Synergy between individual TNF ...... cterium tuberculosis infection
@en
P2093
Denise E Kirschner
JoAnne L Flynn
P2860
P304
P356
10.4049/JIMMUNOL.0802297
P407
P50
P577
2009-03-01T00:00:00Z