Silencing of rotavirus NSP4 or VP7 expression reduces alterations in Ca2+ homeostasis induced by infection of cultured cells.
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Rotavirus vaccines and pathogenesis: 2008Viral Membrane Channels: Role and Function in the Virus Life CycleRotavirus non-structural proteins: structure and functionActivation of the endoplasmic reticulum calcium sensor STIM1 and store-operated calcium entry by rotavirus requires NSP4 viroporin activity.Use of genetically-encoded calcium indicators for live cell calcium imaging and localization in virus-infected cellsExpression of nonstructural rotavirus protein NSP4 mimics Ca2+ homeostasis changes induced by rotavirus infection in cultured cells.Silencing of the rotavirus NSP4 protein decreases the incidence of biliary atresia in murine modelRotavirus disrupts calcium homeostasis by NSP4 viroporin activityRotavirus infection of cells in culture induces activation of RhoA and changes in the actin and tubulin cytoskeletonTubular structure induced by a plant virus facilitates viral spread in its vector insectRotavirus and Serotonin Cross-Talk in DiarrhoeaEngineering and expression of a human rotavirus candidate vaccine in Nicotiana benthamianaToxin mediated diarrhea in the 21 century: the pathophysiology of intestinal ion transport in the course of ETEC, V. cholerae and rotavirus infection.Viruses as modulators of mitochondrial functions.Tyrosine phosphorylation modulates mitochondrial chaperonin Hsp60 and delays rotavirus NSP4-mediated apoptotic signaling in host cells.In vitro antiviral activity of Lactobacillus casei and Bifidobacterium adolescentis against rotavirus infection monitored by NSP4 protein production.The molecular biology of rotaviruses X: intercellular dissemination of rotavirus NSP4 requires glycosylation and is mediated by direct cell-cell contact through cytoplasmic extrusions.Rotavirus infection activates the UPR but modulates its activity.Rotavirus NSP486-175 interacts with H9c2(2-1) cells in vitro, elevates intracellular Ca2+ levels and can become cytotoxic: a possible mechanism for extra-intestinal pathogenesis.The Molecular Switch of Telomere Phages: High Binding Specificity of the PY54 Cro Lytic Repressor to a Single Operator Site.
P2860
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P2860
Silencing of rotavirus NSP4 or VP7 expression reduces alterations in Ca2+ homeostasis induced by infection of cultured cells.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Silencing of rotavirus NSP4 or ...... y infection of cultured cells.
@en
type
label
Silencing of rotavirus NSP4 or ...... y infection of cultured cells.
@en
prefLabel
Silencing of rotavirus NSP4 or ...... y infection of cultured cells.
@en
P2093
P2860
P356
P1433
P1476
Silencing of rotavirus NSP4 or ...... y infection of cultured cells.
@en
P2093
Esmeralda Vizzi
Fabián Michelangeli
Ferdinando Liprandi
Franshelle Peña
José Luis Zambrano
Juan Ernesto Ludert
Marie-Christine Ruiz
Yuleima Díaz
P2860
P304
P356
10.1128/JVI.02719-07
P407
P577
2008-04-09T00:00:00Z