Dissociation of Oct-1 from the nuclear peripheral structure induces the cellular aging-associated collagenase gene expression.
about
Chromatin states and nuclear organization in development--a view from the nuclear laminaPhysiological importance of RNA and protein mobility in the cell nucleusPrelamin A accumulation and stress conditions induce impaired Oct-1 activity and autophagy in prematurely aged human mesenchymal stem cellDynamic regulation of Oct1 during mitosis by phosphorylation and ubiquitination.Maintenance of a functional higher order chromatin structure: The role of the nuclear matrix in normal and disease states.Oct1 is a switchable, bipotential stabilizer of repressed and inducible transcriptional states.Laminopathies: multiple disorders arising from defects in nuclear architecture.Roles of the nuclear lamina in stable nuclear association and assembly of a herpesviral transactivator complex on viral immediate-early genes.Nuclear lamins: key regulators of nuclear structure and activitiesA-type lamins: guardians of the soma?Age-dependent deficiency in import of mitochondrial DNA glycosylases required for repair of oxidatively damaged bases.Accumulation of multipotent progenitors with a basal differentiation bias during aging of human mammary epitheliaNuclear organization of mammalian genomes. Polar chromosome territories build up functionally distinct higher order compartments.Gene silencing at the nuclear periphery.The Oct1 transcription factor and epithelial malignancies: Old protein learns new tricksRegulation of Oct1/Pou2f1 transcription activity by O-GlcNAcylation.The inner nuclear envelope as a transcription factor resting place.Oct transcription factors in development and stem cells: insights and mechanisms.Contributions of extracellular matrix signaling and tissue architecture to nuclear mechanisms and spatial organization of gene expression controlFrom heterochromatin islands to the NAD World: a hierarchical view of aging through the functions of mammalian Sirt1 and systemic NAD biosynthesis.Lamin-B1 contributes to the proper timing of epicardial cell migration and function during embryonic heart development.Aberrant DNA methylation profile in cholangiocarcinoma.Defects in lamin B1 expression or processing affect interphase chromosome position and gene expression.Activation of the 9E3/cCAF chemokine by phorbol esters occurs via multiple signal transduction pathways that converge to MEK1/ERK2 and activate the Elk1 transcription factor.The locus control region is required for association of the murine beta-globin locus with engaged transcription factories during erythroid maturationLamin B1 controls oxidative stress responses via Oct-1.Protein sequestration at the nuclear periphery as a potential regulatory mechanism in premature aging.A study of goat SRY protein expression suggests putative new roles for this gene in the developing testis of a species with long-lasting SRY expression.
P2860
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P2860
Dissociation of Oct-1 from the nuclear peripheral structure induces the cellular aging-associated collagenase gene expression.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Dissociation of Oct-1 from the ...... d collagenase gene expression.
@en
type
label
Dissociation of Oct-1 from the ...... d collagenase gene expression.
@en
prefLabel
Dissociation of Oct-1 from the ...... d collagenase gene expression.
@en
P2093
P2860
P356
P1476
Dissociation of Oct-1 from the ...... d collagenase gene expression.
@en
P2093
M Hasegawa
M Tomifuji
S Nishibayashi
P2860
P304
P356
10.1091/MBC.8.12.2407
P577
1997-12-01T00:00:00Z