TMPRSS2-ERG fusion co-opts master transcription factors and activates NOTCH signaling in primary prostate cancer.

TMPRSS2-ERG fusion co-opts master transcription factors and activates NOTCH signaling in primary prostate cancer.

Item
http://www.wikidata.org/entity/Q38637158
ALK1Fc Suppresses the Human Prostate Cancer Growth in in Vitro and in Vivo Preclinical Models.Somatic Superenhancer Duplications and Hotspot Mutations Lead to Oncogenic Activation of the KLF5 Transcription Factor.Expanding the effects of ERG on chromatin landscapes and dysregulated transcription in prostate cancer.Characterizing steroid hormone receptor chromatin binding landscapes in male and female breast cancer.Multiple roles of Ring 1 and YY1 binding protein in physiology and disease.Delineation of the androgen-regulated signaling pathways in prostate cancer facilitates the development of novel therapeutic approaches.Recent Advances in Prostate Cancer Treatment and Drug Discovery.Integrated Analysis of a Competing Endogenous RNA Network Revealing a Prognostic Signature for Cervical Cancer
P2860

TMPRSS2-ERG fusion co-opts master transcription factors and activates NOTCH signaling in primary prostate cancer.

Item
http://www.wikidata.org/entity/Q38637158
description
2017 nî lūn-bûn
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2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
TMPRSS2-ERG fusion co-opts mas ...... ng in primary prostate cancer.
@en
type
label
TMPRSS2-ERG fusion co-opts mas ...... ng in primary prostate cancer.
@en
prefLabel
TMPRSS2-ERG fusion co-opts mas ...... ng in primary prostate cancer.
@en
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P407
P433
P478
P50
P577
P698
P356
P1433
P1476
TMPRSS2-ERG fusion co-opts mas ...... ing in primary prostate cancer
@en
P2093
P2860
P2888
P304
P31
P356
P407
P433
P478
P50
P577
P698