about
Different patterns of nicotinic acetylcholine receptor subunit transcription in human thymus.Clinical and structural impact of mutations affecting the residue Phe367 of FOXP3 in patients with IPEX syndrome.Reassessing the role of HLA-DRB3 T-cell responses: evidence for significant expression and complementary antigen presentation.Chemokines determine local lymphoneogenesis and a reduction of circulating CXCR4+ T and CCR7 B and T lymphocytes in thyroid autoimmune diseases.Efferocytosis promotes suppressive effects on dendritic cells through prostaglandin E2 production in the context of autoimmunity.Lymphoid neogenesis in chronic inflammatory diseases.The chemokine network. I. How the genomic organization of chemokines contains clues for deciphering their functional complexity.Myelin peptides in multiple sclerosis.Immunological senescence and thymic function in transplantation.Global gene expression changes in type 1 diabetes: insights into autoimmune response in the target organ and in the periphery.Analysis of the cumulative changes in Graves' disease thyroid glands points to IFN signature, plasmacytoid DCs and alternatively activated macrophages as chronicity determining factors.Genetics of Graves' Disease: Special Focus on the Role of TSHR Gene.AIRE genetic variants and predisposition to polygenic autoimmune disease: The case of Graves' disease and a systematic literature review.Population structure in copy number variation and SNPs in the CCL4L chemokine gene.Statin-associated autoimmune myopathy: A distinct new IFL pattern can increase the rate of HMGCR antibody detection by clinical laboratories.[Snake bite poisoning. Review of six cases (author's transl)]Overexpression of metallothionein I/II: a new feature of thyroid follicular cells in Graves' disease.Organ-specific autoimmunity: a 1986 overview.Puzzling diabetic transgenic mice: a lesson for human type 1 diabetes?Inappropriate HLA class II expression on epithelial cells: new insight in the pathogenesis of distinctive autoimmunity.Inappropriate major histocompatibility complex class II expression by thyroid follicular cells in thyroid autoimmune disease and by pancreatic beta cells in type I diabetes.Pancreatic beta-cell damage. In search of novel pathogenetic factors.Epithelial expression of HLA class II molecules: a new pathogenic factor in organ-specific autoimmunity.New ideas in thyroid autoimmunity.Mechanisms of autoimmunity: relevance to the pathogenesis of type I (insulin-dependent) diabetes mellitus.Dendritic cells pulsed with antigen-specific apoptotic bodies prevent experimental type 1 diabetes.Characterisation of the NES2Y cell line and its use in the production of human glucose-responsive insulin producing (hGRIP) cell lines by cell-cell fusion.Peripherin is a relevant neuroendocrine autoantigen recognized by islet-infiltrating B lymphocytes.Reg (regenerating) gene overexpression in islets from non-obese diabetic mice with accelerated diabetes: role of IFNbeta.Primary alloproliferative TH1 response induced by immature plasmacytoid dendritic cells in collaboration with myeloid DCs.HLA-B27 genotyping by fluorescent resonance emission transfer (FRET) probes in real-time PCR.Clinical laboratory standard capillary protein electrophoresis alerted of a low C3 state and lead to the identification of a Factor I deficiency due to a novel homozygous mutation.Novel Mutations Causing C5 Deficiency in Three North-African Families.[Determinación of islet-cell antibodies in different groups of diabetics (author's transl)]Hyperexpression of transporter in antigen processing-1 (TAP-1) in thyroid glands affected by autoimmunity: a contributing factor to the breach of tolerance to thyroid antigens?Self-reactive cytotoxic gamma delta T lymphocytes in Graves' disease specifically recognize thyroid epithelial cells.[Hypotheses on the etiology of insulin-dependent diabetes: published and recent ideas]Graves' disease TSHR-stimulating antibodies (TSAbs) induce the activation of immature thymocytes: a clue to the riddle of TSAbs generation?[Immunopathology of insulin-dependent diabetes. New hypothesis][Autoimmune polyendocrinopathies. Pathogenic hypotheses].
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