EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
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Epstein-Barr Virus LMP1 Mediated Oncogenicity.Host Tumor Suppressor p18INK4c functions as a potent cell-intrinsic inhibitor of γHV68 reactivation and pathogenesis.Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of and the Locus via Interaction with the Histone Lysine Demethylase KDM2B
P2860
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
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2017 nî lūn-bûn
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2017年の論文
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EBV epigenetically suppresses ...... stablishing long-term latency.
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type
label
EBV epigenetically suppresses ...... stablishing long-term latency.
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prefLabel
EBV epigenetically suppresses ...... stablishing long-term latency.
@en
P2093
P2860
P50
P1433
P1476
EBV epigenetically suppresses ...... stablishing long-term latency.
@en
P2093
Gillian A Parker
Kostas Paschos
Quentin Bazot
P2860
P304
P356
10.1371/JOURNAL.PBIO.2001992
P407
P577
2017-08-03T00:00:00Z