An epigenetic switch ensures transposon repression upon dynamic loss of DNA methylation in embryonic stem cells.
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The DNA methyltransferase DNMT3C protects male germ cells from transposon activityReprogramming towards totipotency is greatly facilitated by synergistic effects of small moleculesPolycomb repressive complex 1 provides a molecular explanation for repeat copy number dependency in FSHD muscular dystrophy.Activation of Lineage Regulators and Transposable Elements across a Pluripotent Spectrum.Impairment of DNA Methylation Maintenance Is the Main Cause of Global Demethylation in Naive Embryonic Stem Cells.PRC2 is required for extensive reorganization of H3K27me3 during epigenetic reprogramming in mouse fetal germ cells.Epigenetic resetting of human pluripotency.Capturing Human Naïve Pluripotency in the Embryo and in the Dish.An RB-EZH2 Complex Mediates Silencing of Repetitive DNA Sequences.Vitamin C induces specific demethylation of H3K9me2 in mouse embryonic stem cells via Kdm3a/bMERVL/Zscan4 Network Activation Results in Transient Genome-wide DNA Demethylation of mESCs.DUX-family transcription factors regulate zygotic genome activation in placental mammals.The emergence of piRNAs against transposon invasion to preserve mammalian genome integrity.Rif1 promotes a repressive chromatin state to safeguard against endogenous retrovirus activation.H3K14ac is linked to methylation of H3K9 by the triple Tudor domain of SETDB1.An endosiRNA-Based Repression Mechanism Counteracts Transposon Activation during Global DNA Demethylation in Embryonic Stem Cells.Transient transcription in the early embryo sets an epigenetic state that programs postnatal growth.Genome-wide analyses reveal a role of Polycomb in promoting hypomethylation of DNA methylation valleys.SETDB1 prevents TET2-dependent activation of IAP retroelements in naïve embryonic stem cells.Gene body DNA methylation conspires with H3K36me3 to preclude aberrant transcription.Silencing of transposable elements may not be a major driver of regulatory evolution in primate iPSCs.Transposable elements generate regulatory novelty in a tissue-specific fashion.Transcriptional control and exploitation of an immune-responsive family of plant retrotransposons.L1 retrotransposition in the soma: a field jumping ahead.Dual Inhibition of DNA and Histone Methyltransferases Increases Viral Mimicry in Ovarian Cancer CellsReduced PRC2 function alters male germline epigenetic programming and paternal inheritancePairs of Adjacent Conserved Noncoding Elements Separated by Conserved Genomic Distances Act as Cis-Regulatory Units
P2860
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P2860
An epigenetic switch ensures transposon repression upon dynamic loss of DNA methylation in embryonic stem cells.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
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2016年學術文章
@zh-hant
name
An epigenetic switch ensures t ...... ation in embryonic stem cells.
@en
type
label
An epigenetic switch ensures t ...... ation in embryonic stem cells.
@en
prefLabel
An epigenetic switch ensures t ...... ation in embryonic stem cells.
@en
P2860
P356
P1433
P1476
An epigenetic switch ensures t ...... ation in embryonic stem cells.
@en
P2093
Aurélie Teissandier
Marius Walter
P2860
P356
10.7554/ELIFE.11418
P407
P577
2016-01-27T00:00:00Z