The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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The Combination of Human Urinary Kallidinogenase and Mild Hypothermia Protects Adult Rats Against Hypoxic-Ischemic Encephalopathy-Induced Injury by Promoting Angiogenesis and Regeneration

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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Aiko Inoue

http://www.w3.org/2001/XMLSchema#string

Chang-Ning Hao

http://www.w3.org/2001/XMLSchema#string

Guang Yang

http://www.w3.org/2001/XMLSchema#string

Guangxian Zhao

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Hongxian Wu

http://www.w3.org/2001/XMLSchema#string

Kenji Okumura

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Limei Piao

http://www.w3.org/2001/XMLSchema#string

Lina Hu

http://www.w3.org/2001/XMLSchema#string

Masafumi Kuzuya

http://www.w3.org/2001/XMLSchema#string

Shinyu Ogasawara

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Inhibition of vascular endothelial cell growth factor activity by an endogenously encoded soluble receptor

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

Mechanisms of angiogenesis and arteriogenesis

Mechanisms underlying the impairment of ischemia-induced neovascularization in matrix metalloproteinase 2-deficient mice.

Aging and survival of cutaneous microvasculature.

An antiangiogenic isoform of VEGF-A contributes to impaired vascularization in peripheral artery disease

Exercise training stimulates ischemia-induced neovascularization via phosphatidylinositol 3-kinase/Akt-dependent hypoxia-induced factor-1 alpha reactivation in mice of advanced age

Arterial and cardiac aging: major shareholders in cardiovascular disease enterprises: Part I: aging arteries: a "set up" for vascular disease.

The vascular endothelial growth factor (VEGF) receptor Flt-1 (VEGFR-1) modulates Flk-1 (VEGFR-2) signaling during blood vessel formation

Cathepsin K-mediated Notch1 activation contributes to neovascularization in response to hypoxia.

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287-300

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scholarly article

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10.14336/AD.2016.0920

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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2017-05-02T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28580185

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P921

neovascularization

P932

5440109

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice

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P1476

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P2860

P304

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P356

P433

P478

P577

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P1476

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P478

P577

P698

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