Vitiligo: An Update on Pathophysiology and Treatment Options.

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Matrix metalloproteinase in acral and non-acral vitiligo.Aryl hydrocarbon receptor (AhR) transcription is decreased in skin of vitiligo patients.

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Vitiligo: An Update on Pathophysiology and Treatment Options.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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Vitiligo: An Update on Pathophysiology and Treatment Options.

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Vitiligo: An Update on Pathophysiology and Treatment Options.

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Vitiligo: An Update on Pathophysiology and Treatment Options.

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Vitiligo: An Update on Pathophysiology and Treatment Options

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Nanja van Geel

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P2860

Variant of TYR and autoimmunity susceptibility loci in generalized vitiligo

Genetic Susceptibility to Vitiligo: GWAS Approaches for Identifying Vitiligo Susceptibility Genes and Loci

Genome-wide association analyses identify 13 new susceptibility loci for generalized vitiligo

Increased sensitivity of melanocytes to oxidative stress and abnormal expression of tyrosinase-related protein in vitiligo.

Depigmentation therapies for normal skin in vitiligo universalis.

CXCL10 is critical for the progression and maintenance of depigmentation in a mouse model of vitiligo.

In vivo vitiligo induction and therapy model: double-blind, randomized clinical trial.

New insights in segmental vitiligo: case report and review of theories.

Tacrolimus ointment promotes repigmentation of vitiligo in children: a review of 57 cases.

Tofacitinib Citrate for the Treatment of Vitiligo: A Pathogenesis-Directed Therapy.

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s40257-017-0298-5

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733-744

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scholarly article

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10.1007/S40257-017-0298-5

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Reinhart Speeckaert

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2017-12-01T00:00:00Z

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1085743452

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Vitiligo: An Update on Pathophysiology and Treatment Options.

about

P2860

P2860

Vitiligo: An Update on Pathophysiology and Treatment Options.

description

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type

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P1476

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P2860

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